Literature DB >> 23873971

Very low levels of education and cognitive reserve: a clinicopathologic study.

Jose Marcelo Farfel1, Ricardo Nitrini, Claudia Kimie Suemoto, Lea Tenenholz Grinberg, Renata Eloah Lucena Ferretti, Renata Elaine Paraizo Leite, Edilaine Tampellini, Luzia Lima, Daniela Souza Farias, Ricardo Caires Neves, Roberta Diehl Rodriguez, Paulo Rossi Menezes, Felipe Fregni, David A Bennett, Carlos Augusto Pasqualucci, Wilson Jacob Filho.   

Abstract

OBJECTIVE: We conducted a clinicopathologic study in a large population with very low levels of education to determine whether very few years of education could contribute to cognitive reserve and modify the relation of neuropathologic indices to dementia.
METHODS: In this cross-sectional study, we included 675 individuals 50 years of age or older from the Brazilian Aging Brain Study Group. Cognitive abilities were evaluated through a structured interview with an informant at the time of autopsy, including the Clinical Dementia Rating (CDR) scale. Neuropathologic examinations were performed using immunohistochemistry and following internationally accepted criteria. Multivariate linear regression models were conducted to determine whether the association between cognitive abilities (measured by CDR sum of boxes) and years of education was independent of sociodemographic variables and neuropathologic indices, including neuritic plaques, neurofibrillary tangles, lacunar infarctions, small-vessel disease, and Lewy bodies. In addition, interaction models were used to examine whether education modified the relation between neuropathologic indices and cognition.
RESULTS: Mean education was 3.9 ± 3.5 years. Formal education was associated with a lower CDR sum of boxes (β = -0.197; 95% confidence interval -0.343, -0.052; p = 0.008), after adjustment for sociodemographic variables and neuropathologic indices. Furthermore, education modified the relationship of lacunar infarcts with cognitive abilities (p = 0.04).
CONCLUSIONS: Even a few years of formal education contributes to cognitive reserve.

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Year:  2013        PMID: 23873971      PMCID: PMC3775692          DOI: 10.1212/WNL.0b013e3182a08f1b

Source DB:  PubMed          Journal:  Neurology        ISSN: 0028-3878            Impact factor:   9.910


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