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Literature DB >> 23842645

The DREAM complex: master coordinator of cell cycle-dependent gene expression.

Subhashini Sadasivam¹, James A DeCaprio.

Abstract

The dimerization partner, RB-like, E2F and multi-vulval class B (DREAM) complex provides a previously unsuspected unifying role in the cell cycle by directly linking p130, p107, E2F, BMYB and forkhead box protein M1. DREAM mediates gene repression during the G0 phase and coordinates periodic gene expression with peaks during the G1/S and G2/M phases. Perturbations in DREAM complex regulation shift the balance from quiescence towards proliferation and contribute to the increased mitotic gene expression levels that are frequently observed in cancers with a poor prognosis.

Entities: CellLine Chemical Disease Gene Mutation Species

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Year: 2013 PMID： 23842645 PMCID： PMC3986830 DOI： 10.1038/nrc3556

Source DB: PubMed Journal: Nat Rev Cancer ISSN： 1474-175X Impact factor: 60.716

140 in total

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3. Distinct mechanisms act in concert to mediate cell cycle arrest.

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8. Chromosome-biased binding and gene regulation by the Caenorhabditis elegans DRM complex.

Authors: Tomoko M Tabuchi; Bart Deplancke; Naoki Osato; Lihua J Zhu; M Inmaculada Barrasa; Melissa M Harrison; H Robert Horvitz; Albertha J M Walhout; Kirsten A Hagstrom
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9. E2F-8: an E2F family member with a similar organization of DNA-binding domains to E2F-7.

Authors: Nicola Logan; Anne Graham; Xuijie Zhao; Rebecca Fisher; Baidehi Maiti; Gustavo Leone; Nicholas B La Thangue
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10. LIN54 is an essential core subunit of the DREAM/LINC complex that binds to the cdc2 promoter in a sequence-specific manner.

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Journal: FEBS J Date: 2009-09-02 Impact factor: 5.542

231 in total

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Journal: J Virol Date: 2015-04-15 Impact factor: 5.103

3. Ki-67 Contributes to Normal Cell Cycle Progression and Inactive X Heterochromatin in p21 Checkpoint-Proficient Human Cells.

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Review 4. Sizing up the cell cycle: systems and quantitative approaches in Chlamydomonas.

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5. Melanocyte transformation requires complete loss of all pocket protein function via a mechanism that mitigates the need for MAPK pathway activation.

Authors: I D Tonks; P Mukhopadhyay; W A Schroder; A Sorolla; A W Mould; H Y Handoko; B Ferguson; H K Muller; P Keith; N K Hayward; G J Walker; G F Kay
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