Literature DB >> 23830964

A phase II trial of temsirolimus in men with castration-resistant metastatic prostate cancer.

Andrew J Armstrong1, Tong Shen, Susan Halabi, Gabor Kemeny, Rhonda L Bitting, Patricia Kartcheske, Elizabeth Embree, Karla Morris, Carolyn Winters, Tracy Jaffe, Mark Fleming, Daniel J George.   

Abstract

BACKGROUND: Phosphatase and tensin homologue (PTEN) loss is common in advanced prostate cancer, leading to constitutive activation of the PI3 kinase pathway. Temsirolimus blocks mammalian target of rapamycin (mTOR)/target of rapamycin complex 1 (TORC1), a key signaling node in this pathway; its activity in men with advanced castration-resistant metastatic prostate cancer (mCRPC) is unknown.
METHODS: We conducted a single-arm trial of weekly intravenous temsirolimus administration in men with chemorefractory mCRPC who had ≥ 5 circulating tumor cells (CTCs) at baseline. The primary end point was the change in CTCs at 8 weeks; secondary end points were composite progression-free survival (PFS) (excluding prostate-specific antigen [PSA]), PSA and radiographic response rates, safety, and survival. At PSA/CTC progression, an anti-androgen could be added while continuing temsirolimus.
RESULTS: Eleven patients were accrued out of a planned 20; the trial was stopped prematurely because of lack of efficacy/feasibility. Median age was 61 years, with 55% African-Americans and 36% Caucasian patients. Median baseline PSA level was 390 ng/dL, median baseline number of CTCs was 14 cells; 50% of patients had pain, and 63% had undergone ≥ 2 previous chemotherapy regimens. Median CTC decline was 48% and 3 patients experienced decline in CTCs to < 5. However, 73% of men had a persistently unfavorable number of CTCs (≥ 5) and only 1 patient had a ≥ 30% PSA decline. Median PFS was 1.9 months (95% confidence interval [CI], 0.9-3.1) and median overall survival (OS) was 8.8 months (95% CI, 3.1-15.6). Toxicities included grade 4 hypophosphatemia and central nervous system (CNS) hemorrhage, and frequent grade 3 fatigue, anemia, stomatitis, hypokalemia, weakness, and hyperglycemia.
CONCLUSION: Temsirolimus lacked sufficient clinical activity in men with mCRPC, despite transient CTC improvements in some men. Future studies should focus on combination approaches or novel PI3K pathway inhibitors.
Copyright © 2013 Elsevier Inc. All rights reserved.

Entities:  

Keywords:  Castration-resistant; Circulating tumor cells; Epithelial-mesenchymal transition; Metastatic prostate cancer; N-cadherin; Prostate-specific antigen; Temsirolimus; mTOR

Mesh:

Substances:

Year:  2013        PMID: 23830964     DOI: 10.1016/j.clgc.2013.05.007

Source DB:  PubMed          Journal:  Clin Genitourin Cancer        ISSN: 1558-7673            Impact factor:   2.872


  29 in total

1.  Phase I/II study evaluating the safety and clinical efficacy of temsirolimus and bevacizumab in patients with chemotherapy refractory metastatic castration-resistant prostate cancer.

Authors:  Pedro C Barata; Matthew Cooney; Prateek Mendiratta; Ruby Gupta; Robert Dreicer; Jorge A Garcia
Journal:  Invest New Drugs       Date:  2018-11-07       Impact factor: 3.850

Review 2.  Growth factor and signaling pathways and their relevance to prostate cancer therapeutics.

Authors:  Jocelyn L Wozney; Emmanuel S Antonarakis
Journal:  Cancer Metastasis Rev       Date:  2014-09       Impact factor: 9.264

Review 3.  Androgen pathway resistance in prostate cancer and therapeutic implications.

Authors:  Benjamin L Maughan; Emmanuel S Antonarakis
Journal:  Expert Opin Pharmacother       Date:  2015-06-12       Impact factor: 3.889

4.  Phase II trial of the PI3 kinase inhibitor buparlisib (BKM-120) with or without enzalutamide in men with metastatic castration resistant prostate cancer.

Authors:  Andrew J Armstrong; Susan Halabi; Patrick Healy; Joshi J Alumkal; Carolyn Winters; Julie Kephart; Rhonda L Bitting; Carey Hobbs; Colleen F Soleau; Tomasz M Beer; Rachel Slottke; Kelly Mundy; Evan Y Yu; Daniel J George
Journal:  Eur J Cancer       Date:  2017-05-11       Impact factor: 9.162

Review 5.  Impact of Candidate Genetic Polymorphisms in Prostate Cancer: An Overview.

Authors:  S Salvi; V Conteduca; G Gurioli; D Calistri; V Casadio; U De Giorgi
Journal:  Mol Diagn Ther       Date:  2016-02       Impact factor: 4.074

Review 6.  Hyperglycemia Associated With Targeted Oncologic Treatment: Mechanisms and Management.

Authors:  Jonathan W Goldman; Melody A Mendenhall; Sarah R Rettinger
Journal:  Oncologist       Date:  2016-07-29

7.  Safety and Efficacy of Docetaxel, Bevacizumab, and Everolimus for Castration-resistant Prostate Cancer (CRPC).

Authors:  Mitchell E Gross; Tanya B Dorff; David I Quinn; Patricia M Diaz; Olga O Castellanos; David B Agus
Journal:  Clin Genitourin Cancer       Date:  2017-07-14       Impact factor: 2.872

8.  Circulating tumor cells in prostate cancer diagnosis and monitoring: an appraisal of clinical potential.

Authors:  Giuseppe Galletti; Luigi Portella; Scott T Tagawa; Brian J Kirby; Paraskevi Giannakakou; David M Nanus
Journal:  Mol Diagn Ther       Date:  2014-08       Impact factor: 4.074

9.  The interaction between androgen receptor and PDGF-D in the radiation response of prostate carcinoma.

Authors:  Peter Paximadis; Abdo J Najy; Michael Snyder; Hyeong-Reh Kim
Journal:  Prostate       Date:  2016-01-06       Impact factor: 4.104

Review 10.  mTOR Inhibitors in Castration-Resistant Prostate Cancer: A Systematic Review.

Authors:  Cara M Statz; Sara E Patterson; Susan M Mockus
Journal:  Target Oncol       Date:  2017-02       Impact factor: 4.493

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