Literature DB >> 23630079

Resistant starch from high amylose maize (HAM-RS2) and dietary butyrate reduce abdominal fat by a different apparent mechanism.

Kirk Vidrine1, Jianping Ye, Roy J Martin, Kathleen L McCutcheon, Anne M Raggio, Christine Pelkman, Holiday A Durham, June Zhou, Reshani N Senevirathne, Cathy Williams, Frank Greenway, John Finley, Zhanguo Gao, Felicia Goldsmith, Michael J Keenan.   

Abstract

OBJECTIVE: Obesity is a health concern. Resistant starch (RS) type 2 from high-amylose maize (HAM-RS2) and dietary sodium butyrate (SB) reduce abdominal fat in rodents. RS treatment is associated with increased gut hormones peptide YY (PYY) and glucagon-like peptide 1 (GLP-1), but it is not known if SB increases these hormones. DESIGN AND METHODS: This was investigated in a 2 × 2 rat study with HAM-RS2 (0 or 28% weight) and dietary sodium butyrate (0 and 3.2%) resulting in isocaloric treatments: energy control (EC), sodium butyrate (SB), HAM-RS2 (RS), and the combination (SBRS).
RESULTS: RS and SB reduced abdominal fat and the combination reduced abdominal fat compared to SB and RS. RS was associated with increased fermentation in the cecum. Serum PYY and GLP-1 total were increased with RS treatment. RS treatment was associated with increased cecal butyrate produced from fermentation of RS, but there was no cecal increase for dietary SB.
CONCLUSIONS: SB after its absorption into the blood appears to not affect production of PYY and GLP-1, while butyrate from fermentation in the cecum promotes increased PYY and GLP-1. Future studies with lower doses of RS and SB are warranted and the combination may be beneficial for human health.
Copyright © 2013 The Obesity Society.

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Year:  2013        PMID: 23630079     DOI: 10.1002/oby.20501

Source DB:  PubMed          Journal:  Obesity (Silver Spring)        ISSN: 1930-7381            Impact factor:   5.002


  21 in total

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2.  Sodium butyrate epigenetically modulates high-fat diet-induced skeletal muscle mitochondrial adaptation, obesity and insulin resistance through nucleosome positioning.

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3.  The importance of GLP-1 and PYY in resistant starch's effect on body fat in mice.

Authors:  June Zhou; Roy J Martin; Anne M Raggio; Li Shen; Kathleen McCutcheon; Michael J Keenan
Journal:  Mol Nutr Food Res       Date:  2015-03-02       Impact factor: 5.914

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8.  High amylose resistant starch diet ameliorates oxidative stress, inflammation, and progression of chronic kidney disease.

Authors:  Nosratola D Vaziri; Shu-Man Liu; Wei Ling Lau; Mahyar Khazaeli; Sohrab Nazertehrani; Seyed H Farzaneh; Dorothy A Kieffer; Sean H Adams; Roy J Martin
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9.  Resistant Starch Alters the Microbiota-Gut Brain Axis: Implications for Dietary Modulation of Behavior.

Authors:  Mark Lyte; Ashley Chapel; Joshua M Lyte; Yongfeng Ai; Alexandra Proctor; Jay-Lin Jane; Gregory J Phillips
Journal:  PLoS One       Date:  2016-01-08       Impact factor: 3.240

10.  Saccharomyces boulardii administration changes gut microbiota and reduces hepatic steatosis, low-grade inflammation, and fat mass in obese and type 2 diabetic db/db mice.

Authors:  Amandine Everard; Sébastien Matamoros; Lucie Geurts; Nathalie M Delzenne; Patrice D Cani
Journal:  mBio       Date:  2014-06-10       Impact factor: 7.867

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