Literature DB >> 23593534

Negative association between trunk fat, insulin resistance and skeleton in obese women.

Emanuela A Greco1, Davide Francomano, Rachele Fornari, Chiara Marocco, Carla Lubrano, Vincenza Papa, Francesca Wannenes, Luigi Di Luigi, Lorenzo M Donini, Andrea Lenzi, Antonio Aversa, Silvia Migliaccio.   

Abstract

AIM: To evaluate the potential interference of trunk fat (TF) mass on metabolic and skeletal metabolism.
METHODS: In this cross-sectional study, 340 obese women (mean age: 44.8 ± 14 years; body mass index: 36.0 ± 5.9 kg/m(2)) were included. Patients were evaluated for serum vitamin D, osteocalcin (OSCA), inflammatory markers, lipids, glucose and insulin (homeostasis model assessment of insulin resistance, HOMA-IR) levels, and hormones profile. Moreover, all patients underwent measurements of bone mineral density (BMD; at lumbar and hip site) and body composition (lean mass, total and trunk fat mass) by dual-energy X-ray absorptiometry.
RESULTS: Data showed that: (1) high TF mass was inversely correlated with low BMD both at lumbar (P < 0.001) and hip (P < 0.01) sites and with serum vitamin D (P < 0.0005), OSCA (P < 0.0001) and insulin-like growth factor-1 (IGF-1; P < 0.0001) levels; (2) a positive correlation was found between TF and HOMA-IR (P < 0.01), fibrinogen (P < 0.0001) and erythrocyte sedimentation rate (P < 0.0001); (3) vitamin D levels were directly correlated with IGF-1 (P < 0.0005), lumbar (P < 0.006) and hip (P < 0.01) BMD; and (4) inversely with HOMA-IR (P < 0.001) and fibrinogen (P < 0.0005).Multivariate analysis demonstrated that only vitamin D was independent of TF variable.
CONCLUSION: In obese women, TF negatively correlates with BMD independently from vitamin D levels. Reduced IGF-1 and increased inflammatory markers might be some important determinants that account for this relationship.

Entities:  

Keywords:  Inflammation; Insulin resistance; Obesity; Osteocalcin; Skeleton; Trunk fat; Vitamin D

Year:  2013        PMID: 23593534      PMCID: PMC3627415          DOI: 10.4239/wjd.v4.i2.31

Source DB:  PubMed          Journal:  World J Diabetes        ISSN: 1948-9358


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