Literature DB >> 23541229

The role of Toll-like receptor (TLR) 2 and 9 in renal ischemia and reperfusion injury.

Xingzhi Li1, Zhizhong Yun, Zhaohui Tan, Sanxiang Li, Dong Wang, Kewei Ma, Ning Chi, Junfeng Liu, Feng Chen, Guanxin Gao.   

Abstract

OBJECTIVE: To determine the involvement of Toll-like receptor (TLR) 2, TLR9, and TLR2+9 in renal ischemia/reperfusion (I/R) injury.
METHODS: Mice with gene knock-out of TLR2, TLR9, and TLR2+9 underwent renal I/R injury. Tubular damage, expression of proinflammatory cytokines and chemokines, and kidney dysfunction were evaluated on different days after reperfusion.
RESULTS: Mice deficient in TLR2, but not TLR9, were protected from renal I/R injury with less tubular damage, decreased cytokine production, and lower serum creatinine and urea levels than wild-type mice. TLR2+9 double-deficiency did not have an additional effect on tissue injury compared with TLR2 deficiency alone.
CONCLUSION: These results suggest that activation of TLR2 signaling contributes to the pathogenesis of renal I/R injury, whereas TLR9 may be redundant for the development of this injury.
Copyright © 2013 Elsevier Inc. All rights reserved.

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Year:  2013        PMID: 23541229     DOI: 10.1016/j.urology.2013.02.016

Source DB:  PubMed          Journal:  Urology        ISSN: 0090-4295            Impact factor:   2.649


  9 in total

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  9 in total

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