Literature DB >> 23452164

Prospective association of 25(OH)D with metabolic syndrome.

Sheena Kayaniyil1, Stewart B Harris, Ravi Retnakaran, Reinhold Vieth, Julia A Knight, Hertzel C Gerstein, Bruce A Perkins, Bernard Zinman, Anthony J Hanley.   

Abstract

CONTEXT: Vitamin D may play a role in the aetiology of the metabolic syndrome (MetS), yet the majority of previous studies have been cross-sectional, and the limited number of prospective studies has yielded inconsistent results.
OBJECTIVE: To examine the prospective association of vitamin D [25-hydroxyvitamin D, 25(OH)D] with MetS in a multi-ethnic cohort of adults in Ontario, Canada.
DESIGN: Nondiabetic individuals with pre-existing MetS risk factors were recruited for participation in the PROspective Metabolism and ISlet cell Evaluation (PROMISE) cohort study, a longitudinal study of the determinants of insulin resistance and MetS.
METHODS: Of the 654 participants enrolled at baseline, 489 attended a 3-year follow-up visit. There were 301 participants eligible for the analysis of 25(OH)D with incident MetS (age 49·2 ± 9·3 years old, 75·4% female), after excluding 188 (38·5%) prevalent MetS cases at baseline. Longitudinal change in MetS components was assessed in the entire follow-up cohort.
RESULTS: There were 76 (15·5%) participants who developed MetS over the 3-years of follow-up. Multivariate logistic regression analyses indicated a decreased risk of MetS at follow-up per standard deviation increase in baseline 25(OH)D after adjustment for sociodemographics, season, baseline and change in supplement use and physical activity and insulin resistance (OR = 0·63, 95% CI 0·44-0·90). Multivariate linear regression analyses revealed a significant inverse association of baseline 25(OH)D with fasting glucose at follow-up (β = -0·0005, P = 0·025).
CONCLUSIONS: There was a significant inverse association of baseline 25(OH)D with incident MetS, which may be partly driven by its association with glucose homoeostasis.
© 2013 John Wiley & Sons Ltd.

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Year:  2013        PMID: 23452164     DOI: 10.1111/cen.12190

Source DB:  PubMed          Journal:  Clin Endocrinol (Oxf)        ISSN: 0300-0664            Impact factor:   3.478


  20 in total

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2.  Fasting-Induced Transcription Factors Repress Vitamin D Bioactivation, a Mechanism for Vitamin D Deficiency in Diabetes.

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Authors:  Richard C Strange; Kate E Shipman; Sudarshan Ramachandran
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4.  Effects of Vitamin D Receptor Knockout and Vitamin D Deficiency on Corneal Epithelial Wound Healing and Nerve Density in Diabetic Mice.

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5.  Lipid accumulation product and 25-OH-vitamin D deficiency in type 2 diabetes.

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6.  Deficit of vitamin D in pregnancy and growth and overweight in the offspring.

Authors:  E Morales; A Rodriguez; D Valvi; C Iñiguez; A Esplugues; J Vioque; L S Marina; A Jiménez; M Espada; C R Dehli; A Fernández-Somoano; M Vrijheid; J Sunyer
Journal:  Int J Obes (Lond)       Date:  2014-09-05       Impact factor: 5.095

Review 7.  Obesity and hypovitaminosis D: causality or casualty?

Authors:  Silvia Migliaccio; Andrea Di Nisio; Chiara Mele; Lorenzo Scappaticcio; Silvia Savastano; Annamaria Colao
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Review 8.  Vitamin D insufficiency and insulin resistance in obese adolescents.

Authors:  Catherine A Peterson; Aneesh K Tosh; Anthony M Belenchia
Journal:  Ther Adv Endocrinol Metab       Date:  2014-12       Impact factor: 3.565

Review 9.  Vitamin D and energy homeostasis: of mice and men.

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10.  The Effect of Changing Serum 25-Hydroxyvitamin D Concentrations on Metabolic Syndrome: A Longitudinal Analysis of Participants of a Preventive Health Program.

Authors:  Truong-Minh Pham; John Paul Ekwaru; Solmaz Setayeshgar; Paul J Veugelers
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