Literature DB >> 23160277

Whole exome sequencing identified a novel zinc-finger gene ZNF141 associated with autosomal recessive postaxial polydactyly type A.

Umm-e- Kalsoom1, Eva Klopocki, Naveed Wasif, Muhammad Tariq, Saadullah Khan, Jochen Hecht, Peter Krawitz, Stefan Mundlos, Wasim Ahmad.   

Abstract

BACKGROUND: Postaxial polydactyly (PAP) type A is characterised by well-formed functionally developed 5th digit duplication in hands and/or feet. It is genetically heterogeneous condition, inherited both in autosomal recessive and dominant manners. To date one autosomal recessive and four autosomal dominant loci have been mapped on human chromosomes. In the present study we have investigated a consanguineous Pakistani family segregating autosomal recessive PAP type A to identify the gene responsible for this phenotype.
METHODS: Whole exome sequencing combined with homozygosity mapping and array comparative genomic hybridisation (aCGH) analysis was used to search for a genetic cause of PAP type A in the present study.
RESULTS: Exome sequencing identified a missense mutation (c.1420C>T; p.Thr474Ile) in all the affected individuals of the family, in the gene ZNF141, mapped to the telomeric region on chromosome 4p16.3.
CONCLUSION: This study revealed involvement of a zinc finger gene ZNF141 in causing autosomal recessive PAP type A, which may open up interesting perspectives into the function of this protein in limb development.

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Year:  2012        PMID: 23160277     DOI: 10.1136/jmedgenet-2012-101219

Source DB:  PubMed          Journal:  J Med Genet        ISSN: 0022-2593            Impact factor:   6.318


  17 in total

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4.  Exome sequencing revealed a splice site variant in the IQCE gene underlying post-axial polydactyly type A restricted to lower limb.

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Review 9.  Review of literature: genes related to postaxial polydactyly.

Authors:  Prashant Kumar Verma; Ashraf A El-Harouni
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10.  Identification of the genetic basis of sporadic polydactyly in China by targeted sequencing.

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