Literature DB >> 22931243

SNaPshot Assay in Quantitative Detection of Allelic Nondisjunction in Down Syndrome.

Debarati Ghosh1, Sailesh Gochhait, Disha Banerjee, Anindita Chatterjee, Swagata Sinha, Krishnadas Nandagopal.   

Abstract

AIM: We wished to identify markers associated with allelic nondisjunction in nuclear families with Down syndrome (DS) offspring. Since the GRIK1 and GARS-AIRS-GART genes, mapping to chromosome 21q22.1, may be informative in this regard, we genotyped four single-nucleotide polymorphisms [30952599(A/G) rs363484; 30924733(A/G) rs363506; 34901423(A/G) rs2834235; 34877070(A/G) rs7283354] present in these genes using the SNaPshot(™) assay protocol.
RESULTS: We have reported 30952599(A/G)-rs363484 to be monomorphic in our sample population. Genotyping revealed 35/65 families to be informative for 34877070(A/G)-rs7283354 (GARS-AIRS-GART), whereas only 25/65 and 11/65 are informative for 34901423(A/G)-rs2834235 (GARS-AIRS-GART) and 30924733(A/G)-rs363506 (GRIK1) polymorphisms, respectively. The parent- and stage-of-origin of nondisjunction could be traced in 48/65 families using at least one polymorphic marker. A single trio provided internal validation for assignment of the parent- and stage-of-origin of nondisjunction whereby the nondisjoining alleles were independently identified as G-rs363506, G-rs2834235, and G-rs7283354, respectively. An enhanced ratio of meiosis-I to meiosis-II errors during maternal or paternal meioses accounts for allelic nondisjunction.
CONCLUSIONS: The SNaPshot assay is quantitative and permits multiplexing for detection of allelic nondisjunction. Inclusion of additional informative chromosome 21-specific markers may aid rapid aneuploidy detection, screening, and prenatal counseling of parents at risk of having babies with DS.

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Year:  2012        PMID: 22931243      PMCID: PMC3468158          DOI: 10.1089/gtmb.2012.0083

Source DB:  PubMed          Journal:  Genet Test Mol Biomarkers        ISSN: 1945-0257


  34 in total

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4.  Parental origin, nondisjunction, and recombination of the extra chromosome 21 in Down syndrome: a study in a sample of the Colombian population.

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Journal:  Biomedica       Date:  2007-05-31       Impact factor: 0.935

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6.  Genetic and physical mapping of the GLUR5 glutamate receptor gene on human chromosome 21.

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8.  The genetic architecture of Down syndrome phenotypes revealed by high-resolution analysis of human segmental trisomies.

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Journal:  Proc Natl Acad Sci U S A       Date:  2009-07-13       Impact factor: 11.205

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Authors:  M Y Chou; N Rooke; C W Turck; D L Black
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Review 10.  Aneuploidy: from a physiological mechanism of variance to Down syndrome.

Authors:  Mara Dierssen; Yann Herault; Xavier Estivill
Journal:  Physiol Rev       Date:  2009-07       Impact factor: 37.312

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1.  Combining heterogenous data for prediction of disease related and pharmacogenes.

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