Literature DB >> 22885917

The diagnostic and therapeutic aspects of loss-of-function cardiac sodium channelopathies in children.

Priya Chockalingam1, Sally-Ann B Clur, Johannes M P J Breur, Thomas Kriebel, Thomas Paul, Lukas A Rammeloo, Arthur A M Wilde, Nico A Blom.   

Abstract

BACKGROUND: Loss-of-function sodium channelopathies manifest as a spectrum of diseases including Brugada syndrome (BrS) and cardiac conduction disease.
OBJECTIVE: To analyze the diagnostic and therapeutic aspects of these disorders in children.
METHODS: Patients aged ≤ 16 years with genetically confirmed loss-of-function sodium channelopathies (SCN5A mutation), presenting with cardiac symptoms, positive family history, and/or abnormal electrocardiogram (ECG), were included. Abnormal ECG consisted of type 1 BrS ECG and/or prolonged conduction intervals (PR interval/QRS duration > 98th percentile for age).
RESULTS: Among the cohort (n = 33, age 6 ± 5 years, 58% male subjects, 30% probands), 14 (42%) patients were symptomatic, presenting with syncope (n = 5), palpitations (n = 1), supraventricular arrhythmias (n = 3), aborted cardiac arrest (n = 3), and sudden cardiac death (n = 2). Heart rate was 91 ± 26 beats/min, PR interval 168 ± 35 ms, QRS duration 112 ± 20 ms, and heart-rate corrected QT interval 409 ± 26 ms. Conduction intervals were prolonged in 28 (85%) patients; 6 of these patients also had spontaneous type 1 BrS ECG. Eight fever-associated events occurred in 6 patients; 2 of these were vaccination-related fever episodes. Treatment included aggressive antipyretics during fever in all patients; antiarrhythmic treatment included implantable cardioverter-defibrillator (n = 4), pacemaker (n = 2), and beta-blockers, either alone (n = 3) or in combination with device (n = 2). During follow-up (4 ± 4 years), 2 previously symptomatic patients had monomorphic ventricular tachycardia; there were no deaths.
CONCLUSIONS: Diagnosis of loss-of-function sodium channelopathies in children relies on cardiac symptoms, family history, and ECG. Fever and vaccination are potential arrhythmia triggers; conduction delay is the commonest finding on ECG. Beta-blockers have a role in preventing tachycardia-induced arrhythmias; implantable cardioverter-defibrillator should probably be reserved for severe cases.
Copyright © 2012 Heart Rhythm Society. Published by Elsevier Inc. All rights reserved.

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Year:  2012        PMID: 22885917     DOI: 10.1016/j.hrthm.2012.08.011

Source DB:  PubMed          Journal:  Heart Rhythm        ISSN: 1547-5271            Impact factor:   6.343


  15 in total

1.  Genotype-phenotype relationship and risk stratification in loss-of-function SCN5A mutation carriers.

Authors:  Tomas Robyns; Dieter Nuyens; Bert Vandenberk; Cuno Kuiperi; Anniek Corveleyn; Jeroen Breckpot; Christophe Garweg; Joris Ector; Rik Willems
Journal:  Ann Noninvasive Electrocardiol       Date:  2018-04-30       Impact factor: 1.468

2.  Fever-Induced Brugada Syndrome Is More Common Than Previously Suspected: A Cross-Sectional Study from an Endemic Area.

Authors:  Pattara Rattanawong; Wasawat Vutthikraivit; Attawit Charoensri; Tanawat Jongraksak; Awapa Prombandankul; Napatt Kanjanahattakij; Sakda Rungaramsin; Treechada Wisaratapong; Tachapong Ngarmukos
Journal:  Ann Noninvasive Electrocardiol       Date:  2015-07-14       Impact factor: 1.468

Review 3.  Inherited arrhythmia syndromes leading to sudden cardiac death in the young: a global update and an Indian perspective.

Authors:  Priya Chockalingam; Arthur A Wilde
Journal:  Indian Heart J       Date:  2013-12-17

4.  [Ion channel diseases in children].

Authors:  Christian Wolpert; Ulli Parade; Claudia Herrera-Siklody; Christian Strotmann; Norman Rüb
Journal:  Herzschrittmacherther Elektrophysiol       Date:  2014-08-23

5.  Absence of family history and phenotype-genotype correlation in pediatric Brugada syndrome: more burden to bear in clinical and genetic diagnosis.

Authors:  Houria Daimi; Amel Haj Khelil; Khaldoun Ben Hamda; Amelia Aranega; Jemni B E Chibani; Diego Franco
Journal:  Pediatr Cardiol       Date:  2015-03-11       Impact factor: 1.655

6.  Genetically engineered SCN5A mutant pig hearts exhibit conduction defects and arrhythmias.

Authors:  David S Park; Marina Cerrone; Gregory Morley; Carolina Vasquez; Steven Fowler; Nian Liu; Scott A Bernstein; Fang-Yu Liu; Jie Zhang; Christopher S Rogers; Silvia G Priori; Larry A Chinitz; Glenn I Fishman
Journal:  J Clin Invest       Date:  2014-12-15       Impact factor: 14.808

7.  Bringing home the bacon? The next step in cardiac sodium channelopathies.

Authors:  Arthur A M Wilde; Pieter G Postema
Journal:  J Clin Invest       Date:  2014-12-15       Impact factor: 14.808

8.  Fever-induced Brugada pattern: how common is it and what does it mean?

Authors:  Arnon Adler; Guy Topaz; Karin Heller; David Zeltser; Tami Ohayon; Uri Rozovski; Amir Halkin; Raphael Rosso; Shay Ben-Shachar; Charles Antzelevitch; Sami Viskin
Journal:  Heart Rhythm       Date:  2013-07-19       Impact factor: 6.343

9.  A novel NaV1.5 voltage sensor mutation associated with severe atrial and ventricular arrhythmias.

Authors:  Hong-Gang Wang; Wandi Zhu; Ronald J Kanter; Jonathan R Silva; Christina Honeywell; Robert M Gow; Geoffrey S Pitt
Journal:  J Mol Cell Cardiol       Date:  2016-01-19       Impact factor: 5.000

10.  Monomorphic ventricular tachycardia in Brugada syndrome: True-true but related?

Authors:  Lee L Eckhardt
Journal:  Heart Rhythm       Date:  2015-11-26       Impact factor: 6.343

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