Literature DB >> 22806379

Novel FLNC mutation in a patient with myofibrillar myopathy in combination with late-onset cerebellar ataxia.

Giorgio Tasca1, Zagaa Odgerel, Mauro Monforte, Stefania Aurino, Nigel F Clarke, Leigh B Waddell, Bjarne Udd, Enzo Ricci, Lev G Goldfarb.   

Abstract

INTRODUCTION: Mutations in the gene that encodes filamin C, FLNC, represent a rare cause of a distinctive type of myofibrillar myopathy (MFM).
METHODS: We investigated an Italian patient by means of muscle biopsy, muscle and brain imaging and molecular analysis of MFM genes.
RESULTS: The patient harbored a novel 7256C>T, p.Thr2419Met mutation in exon 44 of FLNC. Clinical, pathological and muscle MRI findings were similar to the previously described filaminopathy cases. This patient had, in addition, cerebellar ataxia with atrophy of cerebellum and vermis evident on brain MRI scan. Extensive screening failed to establish a cause of cerebellar atrophy.
CONCLUSIONS: We report an Italian filaminopathy patient, with a novel mutation in a highly conserved region. This case raises the possibility that the disease spectrum caused by FLNC may include cerebellar dysfunction.
Copyright © 2012 Wiley Periodicals, Inc.

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Year:  2012        PMID: 22806379      PMCID: PMC3400116          DOI: 10.1002/mus.23349

Source DB:  PubMed          Journal:  Muscle Nerve        ISSN: 0148-639X            Impact factor:   3.217


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6.  A mutation in the filamin c gene causes myofibrillar myopathy with lower motor neuron syndrome: a case report.

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7.  Exome sequencing identifies titin mutations causing hereditary myopathy with early respiratory failure (HMERF) in families of diverse ethnic origins.

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