Literature DB >> 22613000

Insulin resistance undermines the advantages of IL28B polymorphism in the pegylated interferon alpha-2b and ribavirin treatment of chronic hepatitis C patients with genotype 1.

Eiichi Ogawa1, Norihiro Furusyo, Masayuki Murata, Hiroaki Ikezaki, Takeshi Ihara, Takeo Hayashi, Kazuhiro Toyoda, Hiroaki Taniai, Kyoko Okada, Mosaburo Kainuma, Jun Hayashi.   

Abstract

BACKGROUND & AIMS: Recent studies have suggested that insulin resistance exerts a strong influence on chronic hepatitis C virus (HCV) infection. We analyzed pretreatment factors useful for predicting sustained virological response (SVR), especially interleukin (IL) 28B polymorphism and Homeostasis Model Assessment of Insulin Resistance (HOMA-IR).
METHODS: This cohort study consisted of 328 chronic hepatitis C patients with HCV genotype 1 who were treated for 48 weeks with pegylated interferon (PegIFN) α-2b and ribavirin (RBV). Genotyping of the polymorphisms in the IL28B gene region (rs8099917) on chromosome 19 was performed on DNA collected from each patient.
RESULTS: No significant difference in IL28B genotype distribution was found according to HOMA-IR. Multivariate analysis identified the IL28B TT genotype (OR=5.97, 95% CI 2.15-16.55, p=0.0006) and the baseline HOMA-IR (OR=0.65, 95% CI 0.48-0.87, p=0.0044) as significant, independent pretreatment predictors of SVR. Receiver operating characteristic analyses to determine the optimal threshold values of HOMA-IR for predicting SVR showed that the areas under the curve (AUC) were high for both IL28B TT (AUC=0.774, HOMA-IR cut-off value: 2.45) and IL28B TG/GG genotypes (AUC=0.772, HOMA-IR cut-off value: 1.55).
CONCLUSIONS: For HCV genotype 1, both IL28B and baseline HOMA-IR are independent pretreatment predictors of SVR in patients treated with PegIFNα-2b and RBV. Insulin resistance undermines the advantages of IL28B polymorphism to obtain SVR.
Copyright © 2012 European Association for the Study of the Liver. Published by Elsevier B.V. All rights reserved.

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Year:  2012        PMID: 22613000     DOI: 10.1016/j.jhep.2012.04.027

Source DB:  PubMed          Journal:  J Hepatol        ISSN: 0168-8278            Impact factor:   25.083


  11 in total

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Review 2.  Interleukin 28B polymorphisms as predictors of sustained virological response in chronic hepatitis C: systematic review and meta-analysis.

Authors:  E Cariani; L Roli; G Missale; E Villa; C Ferrari; T Trenti
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Review 4.  Individualization of chronic hepatitis C treatment according to the host characteristics.

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5.  Interaction of IFNL3 with insulin resistance, steatosis and lipid metabolism in chronic hepatitis C virus infection.

Authors:  Mohammed Eslam; David R Booth; Jacob George; Golo Ahlenstiel
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6.  Meta-analysis: implications of interleukin-28B polymorphisms in spontaneous and treatment-related clearance for patients with hepatitis C.

Authors:  María A Jiménez-Sousa; Amanda Fernández-Rodríguez; María Guzmán-Fulgencio; Mónica García-Álvarez; Salvador Resino
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7.  Interferon- α -Induced Changes to Natural Killer Cells Are Associated with the Treatment Outcomes in Patients with HCV Infections.

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Journal:  Hepat Res Treat       Date:  2013-08-13

8.  Pretreatment Predictors of Response to PegIFN-RBV Therapy in Egyptian Patients with HCV Genotype 4.

Authors:  Hanan H Rizk; Nadia M Hamdy; Nadia L Al-Ansari; Hala O El-Mesallamy
Journal:  PLoS One       Date:  2016-04-21       Impact factor: 3.240

9.  Influence of insulin resistance on the development of hepatocellular carcinoma after antiviral treatment for non-cirrhotic patients with chronic hepatitis C.

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Journal:  Infect Agent Cancer       Date:  2016-02-24       Impact factor: 2.965

10.  Revisiting the stopping rule for hepatitis C genotype 1 patients treated with peginterferon plus ribavirin.

Authors:  Ming-Lung Yu; Chen-Hua Liu; Chung-Feng Huang; Tai-Chung Tseng; Jee-Fu Huang; Chia-Yen Dai; Zu-Yau Lin; Shinn-Cherng Chen; Liang-Yen Wang; Suh-Hang Hank Juo; Wan-Long Chuang; Jia-Horng Kao
Journal:  PLoS One       Date:  2012-12-21       Impact factor: 3.240

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