Literature DB >> 22609385

Increased risk for persistent intestinal metaplasia in patients with Barrett's esophagus and uncontrolled reflux exposure before radiofrequency ablation.

Kumar Krishnan1, John E Pandolfino2, Peter J Kahrilas2, Laurie Keefer1, Lubomyr Boris3, Srinadh Komanduri4.   

Abstract

BACKGROUND & AIMS: Radiofrequency ablation (RFA) is a safe alternative to esophagectomy for patients with dysplastic Barrett's esophagus (BE). Although some studies have indicated that RFA is effective at eradicating dysplasia, most have found that RFA is not as effective in eradicating intestinal metaplasia. We investigated whether uncontrolled reflux is associated with persistent intestinal metaplasia after RFA.
METHODS: Thirty-seven patients with BE underwent RFA, high-resolution manometry, and 24-hour impedance-pH testing; they received proton pump inhibitors twice daily. Patients returned every 2 months for repeat treatment or standard surveillance. Patients were classified as complete responders (CRs) if all intestinal metaplasia was eradicated in fewer than 3 ablation sessions. We analyzed clinical parameters to identify factors associated with a CR or incomplete responder (ICR).
RESULTS: Among the 37 patients, 22 had a CR and 15 had an ICR. Mann-Whitney U tests revealed that length of BE, size of hiatal hernia, and frequency of reflux, but not acid reflux, differed between CRs and ICRs. CRs had fewer weakly acidic events than ICRs (29.5 vs 52; P < .05) and total reflux events (33.5 vs 60; P < .05), and a trend toward fewer weakly alkaline events (1.0 vs 5.0; P = .06). No other clinical or manometric features differed between groups.
CONCLUSIONS: Uncontrolled, predominantly weakly acidic reflux despite twice-daily proton pump inhibitor therapy before RFA increases the incidence of persistent intestinal metaplasia after ablation in patients with BE. Length of BE and size of hiatal hernia also were associated with persistent intestinal metaplasia after RFA.
Copyright © 2012 AGA Institute. Published by Elsevier Inc. All rights reserved.

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Year:  2012        PMID: 22609385      PMCID: PMC3429717          DOI: 10.1053/j.gastro.2012.05.005

Source DB:  PubMed          Journal:  Gastroenterology        ISSN: 0016-5085            Impact factor:   22.682


  38 in total

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2.  Oesophageal and gastric pH profiles in patients with gastro-oesophageal reflux disease and Barrett's oesophagus treated with proton pump inhibitors.

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3.  Evaluation of the magnitude of gastro-oesophageal reflux in Barrett's oesophagus.

Authors:  P Parrilla; A Ortiz; L F Martinez de Haro; J L Aguayo; P Ramirez
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6.  Complications of gastroesophageal reflux disease. Role of the lower esophageal sphincter, esophageal acid and acid/alkaline exposure, and duodenogastric reflux.

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8.  Successful elimination of reflux symptoms does not insure adequate control of acid reflux in patients with Barrett's esophagus.

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9.  Determinants of gastroesophageal junction incompetence: hiatal hernia, lower esophageal sphincter, or both?

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10.  Significance of Nissen fundoplication after endoscopic radiofrequency ablation of Barrett's esophagus.

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