Literature DB >> 22505251

Soluble TWEAK level: is it a marker for cardiovascular disease in long-term hemodialysis patients?

Ozkan Gungor1, Fatih Kircelli, Gulay Asci, Juan Jesus Carrero, Erhan Tatar, Meltem Sezis Demirci, Süreyya Süha Ozbek, Naim Ceylan, Huseyin Toz, Mehmet Ozkahya, Ercan Ok.   

Abstract

BACKGROUND: Reduced soluble tumor necrosis factor-like weak inducer of apoptosis (sTWEAK) levels follow declining renal function, are strongly associated with endothelial dysfunction and predict cardiovascular events in nondialyzed chronic kidney disease patients. In contrast, elevated levels of sTWEAK predict poor survival in hemodialysis (HD) patients. Recent evidence suggests a role for sTWEAK in the pathophysiology of vascular calcification. The aim of the study was to investigate plausible links between sTWEAK, atherosclerosis, arterial stiffness and vascular calcification in HD patients.
METHODS: Coronary artery calcification score (CACs) determined by multislice computed tomography, arterial stiffness by pulse wave velocity (PWV) and carotid artery intima-media thickness (CA-IMT) by carotid Doppler ultrasonography were assessed in 131 long-term prevalent HD patients. sTWEAK levels were measured by ELISA (Bender MedSystems, Vienna, Austria).
RESULTS: Mean serum sTWEAK level was 237.0 ± 147.5 pg/mL (range 78-937). sTWEAK level was inversely correlated with CA-IMT at a borderline significance (r=-0.168, p=0.05). Neither carotid-radial PWV nor carotid-femoral PWV values correlated with sTWEAK. sTWEAK level was higher in patients with severe vascular calcification (CACs ≥400) compared to patients with CACs <400 (264.5 ± 146.8 pg/mL vs. 205.04 ± 122.4 pg/mL, p=0.02).The association between sTWEAK and vascular calcification persisted after multivariate adjustment.
CONCLUSIONS: There exists a weak inverse correlation between sTWEAK and carotid atherosclerosis and a positive correlation with coronary artery calcification in long-term HD patients. Our data give support for a role for sTWEAK in the pathogenesis of vascular injury in HD patients.

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Year:  2013        PMID: 22505251     DOI: 10.5301/jn.5000121

Source DB:  PubMed          Journal:  J Nephrol        ISSN: 1121-8428            Impact factor:   3.902


  4 in total

1.  A panel of biomarkers is associated with increased risk of the presence and progression of atherosclerosis in women with systemic lupus erythematosus.

Authors:  Maureen McMahon; Brian J Skaggs; Jennifer M Grossman; Lori Sahakian; John Fitzgerald; Weng Kee Wong; Elaine V Lourenco; Nagesh Ragavendra; Christina Charles-Schoeman; Alan Gorn; George A Karpouzas; Mihaela B Taylor; Karol E Watson; Michael H Weisman; Daniel J Wallace; Bevra H Hahn
Journal:  Arthritis Rheumatol       Date:  2014-01       Impact factor: 10.995

2.  The association of high sCD163/sTWEAK ratio with cardiovascular disease in hemodialysis patients.

Authors:  Crina Claudia Rusu; Simona Racasan; Ina Maria Kacso; Liviu Ghervan; Diana Moldovan; Alina Potra; Ioan Mihai Patiu; Cosmina Bondor; Mirela Gherman Caprioara
Journal:  Int Urol Nephrol       Date:  2015-10-03       Impact factor: 2.370

3.  Soluble TWEAK levels are independently associated with coronary artery disease severity in patients with stage 2-3 kidney disease.

Authors:  Alper Azak; Mehmet Fatih Akdoğan; Nazım Denizli; Bülent Huddam; Gülay Koçak; Murat Gücün; Mustafa Adem Tatlısu; Recep Demirci; Bilal Yılmaz; Mehmet Dikeç; Murat Bakırtaş; Ibrahim Akdağ; Murat Duranay
Journal:  Int Urol Nephrol       Date:  2013-09-17       Impact factor: 2.370

4.  TWEAK favors phosphate-induced calcification of vascular smooth muscle cells through canonical and non-canonical activation of NFκB.

Authors:  L Hénaut; A B Sanz; D Martin-Sanchez; S Carrasco; R Villa-Bellosta; G Aldamiz-Echevarria; Z A Massy; M D Sanchez-Nino; A Ortiz
Journal:  Cell Death Dis       Date:  2016-07-21       Impact factor: 8.469

  4 in total

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