Literature DB >> 26433887

The association of high sCD163/sTWEAK ratio with cardiovascular disease in hemodialysis patients.

Crina Claudia Rusu1, Simona Racasan2, Ina Maria Kacso3, Liviu Ghervan4, Diana Moldovan3, Alina Potra3, Ioan Mihai Patiu2, Cosmina Bondor5, Mirela Gherman Caprioara3.   

Abstract

PURPOSE: Cardiovascular disease (CVD) is the most common cause of death in hemodialysis (HD) patients. Transmembrane proteins that circulate as soluble form such as tumor necrosis factor (TNF)-like weak inducer of apoptosis (TWEAK) and CD163 have been proposed in previous studies as CVD biomarkers in chronic kidney disease patients. In HD patients, since studies are scarce, the role of these proteins is not completely understood. We tested the hypothesis that sTWEAK, sCD163 or sCD163/sTWEAK ratio could be associated with cardiovascular disease in HD patients.
METHODS: We recorded current clinical and biological data, and we measured sTWEAK and sCD163 serum levels by ELISA in 70 hemodialysis patients. Univariate analysis and multivariate (logistic regression) analysis were used to identify the relation between sTWEAK, sCD163 and sCD163/sTWEAK ratio and CVD.
RESULTS: In univariate analysis, CVD in HD patients is associated with higher sCD163/sTWEAK ratio (p = 0.04), sCD163 (p = 0.07), CRP (p = 0.04), age (p = 0.07), smoking (p = 0.09) and vascular calcifications (p = 0.10). In multivariate analysis, only logarithm of sCD163/sTWEAK ratio (p = 0.04) and smoking (p = 0.03) was significantly associated with CVD. The levels of these molecules and their ratio were correlated with atherosclerosis risk factors: diabetes mellitus, high fasting glucose, tricipital skinfold thickness and CRP as well as (for sCD163/sTWEAK) intravenous iron therapy.
CONCLUSIONS: Cardiovascular disease is associated with increased sCD163/sTWEAK ratio. To our knowledge, this is the first report about this relationship in HD patients.

Entities:  

Keywords:  Atherosclerosis; Cardiovascular disease; Hemodialysis; sCD163/sTWEAK ratio

Mesh:

Substances:

Year:  2015        PMID: 26433887     DOI: 10.1007/s11255-015-1114-x

Source DB:  PubMed          Journal:  Int Urol Nephrol        ISSN: 0301-1623            Impact factor:   2.370


  35 in total

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Authors:  Holger J Møller
Journal:  Scand J Clin Lab Invest       Date:  2011-11-07       Impact factor: 1.713

2.  Identification of the hemoglobin scavenger receptor/CD163 as a natural soluble protein in plasma.

Authors:  Holger Jon Møller; Niels Anker Peterslund; Jonas Heilskov Graversen; Søren Kragh Moestrup
Journal:  Blood       Date:  2002-01-01       Impact factor: 22.113

3.  Impact of soluble TWEAK and CD163/TWEAK ratio on long-term cardiovascular mortality in patients with peripheral arterial disease.

Authors:  Grazina Urbonaviciene; Jose L Martin-Ventura; Jes S Lindholt; Sigitas Urbonavicius; Juan A Moreno; Jesús Egido; Luis M Blanco-Colio
Journal:  Atherosclerosis       Date:  2011-09-16       Impact factor: 5.162

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Authors:  Jonas Axelsson; Holger Jon Møller; Anna Witasp; Abdul Rashid Qureshi; Juan Jesus Carrero; Olof Heimbürger; Peter Bárány; Anders Alvestrand; Bengt Lindholm; Søren K Moestrup; Peter Stenvinkel
Journal:  Am J Kidney Dis       Date:  2006-12       Impact factor: 8.860

5.  Soluble TWEAK levels are independently associated with coronary artery disease severity in patients with stage 2-3 kidney disease.

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Journal:  Int Urol Nephrol       Date:  2013-09-17       Impact factor: 2.370

6.  Nutritional status evaluation and survival in haemodialysis patients in one centre from Romania.

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8.  Serum levels of the atherosclerosis biomarker sTWEAK are decreased in type 2 diabetes and end-stage renal disease.

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9.  Reduced circulating sTWEAK levels are associated with metabolic syndrome in elderly individuals at high cardiovascular risk.

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Review 10.  Medial vascular calcification revisited: review and perspectives.

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Journal:  Eur Heart J       Date:  2014-04-16       Impact factor: 29.983

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1.  The metabolic hormone FGF21 is associated with endothelial dysfunction in hemodialysis patients.

Authors:  Crina Claudia Rusu; Simona Racasan; Ina Maria Kacso; Diana Moldovan; Alina Potra; Dacian Tirinescu; Cristian Budurea; Remus Orasan; Ioan Mihai Patiu; Cosmina Ioana Bondor; Dan Vladutiu; Mirela Gherman Caprioara
Journal:  Int Urol Nephrol       Date:  2016-12-10       Impact factor: 2.370

Review 2.  The Pathogenesis of End-Stage Renal Disease from the Standpoint of the Theory of General Pathological Processes of Inflammation.

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