Literature DB >> 22415583

Increased urinary podocytes following indomethacin suggests drug-induced glomerular injury.

Alison L Kent1, Linda Brown, Margaret Broom, Amy Broomfield, Jane E Dahlstrom.   

Abstract

BACKGROUND: Preterm infants are delivered while glomerulogenesis is ongoing and may be exposed to insults, including medications that may affect renal development. Podocytes detected in the urine are an indicator of glomerular injury. The aims of this study were to determine whether preterm and term infants excrete podocytes in their urine and whether exposure to gentamicin and indomethacin increase podocyte excretion in their urine.
METHODS: Preterm infants <33 weeks gestation had urine collected each day while receiving either gentamicin or indomethacin. Preterm and term control infants had urine collected for 3 days. The number of casts and podocytes present in the urine of infants receiving indomethacin and gentamicin were compared with preterm and term control infants.
RESULTS: Forty-two neonates were included in the study. Podocytes were present in small numbers (< 2) in the urine of both preterm and term control neonates. The number of podocytes in the preterm group receiving indomethacin was significantly higher than in all other groups (p=0.02) ,as was urinary albumin (p=0.02).
CONCLUSIONS: Increased number of podocytes in preterm neonates receiving indomethacin and higher excretion of albumin suggest glomerular injury is occurring. It is unknown whether injury to glomeruli during glomerulogenesis in preterm neonates has long-term sequelae for renal development and function into adulthood.

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Year:  2012        PMID: 22415583     DOI: 10.1007/s00467-012-2111-2

Source DB:  PubMed          Journal:  Pediatr Nephrol        ISSN: 0931-041X            Impact factor:   3.714


  46 in total

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2.  Histomorphometric analysis of postnatal glomerulogenesis in extremely preterm infants.

Authors:  Maria M Rodríguez; Alexander H Gómez; Carolyn L Abitbol; Jayanthi J Chandar; Shahnaz Duara; Gastón E Zilleruelo
Journal:  Pediatr Dev Pathol       Date:  2004 Jan-Feb

3.  Urinary excretion of podocytes in patients with diabetic nephropathy.

Authors:  T Nakamura; C Ushiyama; S Suzuki; M Hara; N Shimada; I Ebihara; H Koide
Journal:  Nephrol Dial Transplant       Date:  2000-09       Impact factor: 5.992

4.  Relationship between weight at birth and the number and size of renal glomeruli in humans: a histomorphometric study.

Authors:  R Mañalich; L Reyes; M Herrera; C Melendi; I Fundora
Journal:  Kidney Int       Date:  2000-08       Impact factor: 10.612

5.  Nephron endowment and renal filtration surface area in young spontaneously hypertensive rats.

Authors:  M Jane Black; Yao Wang; John F Bertram
Journal:  Kidney Blood Press Res       Date:  2002       Impact factor: 2.687

6.  Number and size of renal glomeruli in spontaneously hypertensive rats.

Authors:  K Skov; J R Nyengaard; N Korsgaard; M J Mulvany
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Review 7.  Hereditary nephrotic syndrome: a systematic approach for genetic testing and a review of associated podocyte gene mutations.

Authors:  Geneviève Benoit; Eduardo Machuca; Corinne Antignac
Journal:  Pediatr Nephrol       Date:  2010-03-24       Impact factor: 3.714

8.  Indomethacin and renal impairment in neonates.

Authors:  Satoshi Akima; Alison Kent; Graham J Reynolds; Martin Gallagher; Michael C Falk
Journal:  Pediatr Nephrol       Date:  2004-03-09       Impact factor: 3.714

Review 9.  Proteinuria: an enzymatic disease of the podocyte?

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Journal:  Kidney Int       Date:  2009-11-18       Impact factor: 10.612

10.  Renal glomeruli and tubular injury following indomethacin, ibuprofen, and gentamicin exposure in a neonatal rat model.

Authors:  Alison L Kent; Lesley E Maxwell; Mark E Koina; Michael C Falk; David Willenborg; Jane E Dahlstrom
Journal:  Pediatr Res       Date:  2007-09       Impact factor: 3.756

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4.  Urinary Neutrophil Gelatinase-associated Lipocalin in the evaluation of Patent Ductus Arteriosus and AKI in Very Preterm Neonates: a cohort study.

Authors:  Anna Sellmer; Bodil H Bech; Jesper V Bjerre; Michael R Schmidt; Vibeke E Hjortdal; Gitte Esberg; Søren Rittig; Tine B Henriksen
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5.  Increasing urinary podocyte mRNA excretion and progressive podocyte loss in kidney contribute to the high risk of long-term renal disease caused by preterm birth.

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Review 6.  Traditional Chinese Medicine in Treating Primary Podocytosis: From Fundamental Science to Clinical Research.

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