Literature DB >> 22013180

Quantifying physical decline in juvenile neuronal ceroid lipofuscinosis (Batten disease).

J M Kwon1, H Adams, P G Rothberg, E F Augustine, F J Marshall, E A Deblieck, A Vierhile, C A Beck, N J Newhouse, J Cialone, E Levy, D Ramirez-Montealegre, L S Dure, K R Rose, J W Mink.   

Abstract

OBJECTIVE: To use the Unified Batten Disease Rating Scale (UBDRS) to measure the rate of decline in physical and functional capability domains in patients with juvenile neuronal ceroid lipofuscinosis (JNCL) or Batten disease, a neurodegenerative lysosomal storage disorder. We have evaluated the UBDRS in subjects with JNCL since 2002; during that time, the scale has been refined to improve reliability and validity. Now that therapies are being proposed to prevent, slow, or reverse the course of JNCL, the UBDRS will play an important role in quantitatively assessing clinical outcomes in research trials.
METHODS: We administered the UBDRS to 82 subjects with JNCL genetically confirmed by CLN3 mutational analysis. Forty-four subjects were seen for more than one annual visit. From these data, the rate of physical impairment over time was quantified using multivariate linear regression and repeated-measures analysis.
RESULTS: The UBDRS Physical Impairment subscale shows worsening over time that proceeds at a quantifiable linear rate in the years following initial onset of clinical symptoms. This deterioration correlates with functional capability and is not influenced by CLN3 genotype.
CONCLUSION: The UBDRS is a reliable and valid instrument that measures clinical progression in JNCL. Our data support the use of the UBDRS to quantify the rate of progression of physical impairment in subjects with JNCL in clinical trials.

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Year:  2011        PMID: 22013180      PMCID: PMC3233207          DOI: 10.1212/WNL.0b013e318237f649

Source DB:  PubMed          Journal:  Neurology        ISSN: 0028-3878            Impact factor:   9.910


  21 in total

Review 1.  Neurology of the neuronal ceroid-lipofuscinoses: late infantile and juvenile types.

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2.  Linear clinical progression, independent of age of onset, in Niemann-Pick disease, type C.

Authors:  Nicole M Yanjanin; Jorge I Vélez; Andrea Gropman; Kelly King; Simona E Bianconi; Sandra K Conley; Carmen C Brewer; Beth Solomon; William J Pavan; Mauricio Arcos-Burgos; Marc C Patterson; Forbes D Porter
Journal:  Am J Med Genet B Neuropsychiatr Genet       Date:  2010-01-05       Impact factor: 3.568

3.  Can focusing on UPDRS Part II make assessments of Parkinson disease progression more efficient?

Authors:  Cristina Sampaio
Journal:  Nat Clin Pract Neurol       Date:  2009-03

4.  Delayed classic and protracted phenotypes of compound heterozygous juvenile neuronal ceroid lipofuscinosis.

Authors:  L Lauronen; P B Munroe; I Järvelä; T Autti; H M Mitchison; A M O'Rawe; R M Gardiner; S E Mole; J Puranen; A M Häkkinen; E Kirveskari; P Santavuori
Journal:  Neurology       Date:  1999-01-15       Impact factor: 9.910

5.  A clinical rating scale for Batten disease: reliable and relevant for clinical trials.

Authors:  F J Marshall; E A de Blieck; J W Mink; L Dure; H Adams; S Messing; P G Rothberg; E Levy; T McDonough; J DeYoung; M Wang; D Ramirez-Montealegre; J M Kwon; D A Pearce
Journal:  Neurology       Date:  2005-07-26       Impact factor: 9.910

6.  Compound heterozygous genotype is associated with protracted juvenile neuronal ceroid lipofuscinosis.

Authors:  K E Wisniewski; N Zhong; W Kaczmarski; A Kaczmarski; E Kida; W T Brown; K O Schwarz; A M Lazzarini; A J Rubin; E S Stenroos; W G Johnson; T M Wisniewski
Journal:  Ann Neurol       Date:  1998-01       Impact factor: 10.422

7.  Genomic structure and complete nucleotide sequence of the Batten disease gene, CLN3.

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Journal:  Genomics       Date:  1997-03-01       Impact factor: 5.736

8.  Isolation of a novel gene underlying Batten disease, CLN3. The International Batten Disease Consortium.

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Journal:  Cell       Date:  1995-09-22       Impact factor: 41.582

Review 9.  Current state of clinical and morphological features in human NCL.

Authors:  Hans H Goebel; Krystyna E Wisniewski
Journal:  Brain Pathol       Date:  2004-01       Impact factor: 6.508

Review 10.  CLN3, the protein associated with batten disease: structure, function and localization.

Authors:  Seasson N Phillips; Jared W Benedict; Jill M Weimer; David A Pearce
Journal:  J Neurosci Res       Date:  2005-03-01       Impact factor: 4.433

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  21 in total

1.  Females experience a more severe disease course in Batten disease.

Authors:  Jennifer Cialone; Heather Adams; Erika F Augustine; Frederick J Marshall; Jennifer M Kwon; Nicole Newhouse; Amy Vierhile; Erika Levy; Leon S Dure; Katherine R Rose; Denia Ramirez-Montealegre; Elisabeth A de Blieck; Jonathan W Mink
Journal:  J Inherit Metab Dis       Date:  2011-12-14       Impact factor: 4.982

2.  Quantitative telemedicine ratings in Batten disease: implications for rare disease research.

Authors:  J Cialone; E F Augustine; N Newhouse; A Vierhile; F J Marshall; J W Mink
Journal:  Neurology       Date:  2011-10-19       Impact factor: 9.910

Review 3.  Classification and natural history of the neuronal ceroid lipofuscinoses.

Authors:  Jonathan W Mink; Erika F Augustine; Heather R Adams; Frederick J Marshall; Jennifer M Kwon
Journal:  J Child Neurol       Date:  2013-07-09       Impact factor: 1.987

4.  Clinical trials in rare disease: challenges and opportunities.

Authors:  Erika F Augustine; Heather R Adams; Jonathan W Mink
Journal:  J Child Neurol       Date:  2013-09       Impact factor: 1.987

5.  A Case-controlled Investigation of Pain Experience and Sensory Function in Neuronal Ceroid Lipofuscinosis.

Authors:  Chantel C Barney; John Hoch; Breanne Byiers; Adele Dimian; Frank J Symons
Journal:  Clin J Pain       Date:  2015-11       Impact factor: 3.442

6.  Methodology of clinical research in rare diseases: development of a research program in juvenile neuronal ceroid lipofuscinosis (JNCL) via creation of a patient registry and collaboration with patient advocates.

Authors:  Elisabeth A de Blieck; Erika F Augustine; Frederick J Marshall; Heather Adams; Jennifer Cialone; Leon Dure; Jennifer M Kwon; Nicole Newhouse; Katherine Rose; Paul G Rothberg; Amy Vierhile; Jonathan W Mink
Journal:  Contemp Clin Trials       Date:  2013-04-26       Impact factor: 2.226

Review 7.  Future perspectives: Moving towards NCL treatments.

Authors:  Susan L Cotman; Sara E Mole; Romina Kohan
Journal:  Biochim Biophys Acta       Date:  2015-04-07

8.  Short-Term Administration of Mycophenolate Is Well-Tolerated in CLN3 Disease (Juvenile Neuronal Ceroid Lipofuscinosis).

Authors:  Erika F Augustine; Christopher A Beck; Heather R Adams; Sara Defendorf; Amy Vierhile; Derek Timm; Jill M Weimer; Jonathan W Mink; Frederick J Marshall
Journal:  JIMD Rep       Date:  2018-06-20

9.  Standardized assessment of seizures in patients with juvenile neuronal ceroid lipofuscinosis.

Authors:  Erika F Augustine; Heather R Adams; Christopher A Beck; Amy Vierhile; Jennifer Kwon; Paul G Rothberg; Frederick Marshall; Robert Block; James Dolan; Jonathan W Mink
Journal:  Dev Med Child Neurol       Date:  2014-11-11       Impact factor: 5.449

10.  Role of the Lysosomal Membrane Protein, CLN3, in the Regulation of Cathepsin D Activity.

Authors:  Jaime Cárcel-Trullols; Attila D Kovács; David A Pearce
Journal:  J Cell Biochem       Date:  2017-05-23       Impact factor: 4.429

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