Literature DB >> 25387857

Standardized assessment of seizures in patients with juvenile neuronal ceroid lipofuscinosis.

Erika F Augustine1, Heather R Adams, Christopher A Beck, Amy Vierhile, Jennifer Kwon, Paul G Rothberg, Frederick Marshall, Robert Block, James Dolan, Jonathan W Mink.   

Abstract

AIM: To evaluate seizure phenomenology, treatment, and course in individuals with juvenile neuronal ceroid lipofuscinosis (JNCL).
METHOD: Data from an ongoing natural history study of JNCL were analyzed using cross-sectional and longitudinal methods. Seizures were evaluated with the Unified Batten Disease Rating Scale, a disease-specific quantitative assessment tool.
RESULTS: Eighty-six children (44 males, 42 females) with JNCL were assessed at an average of three annual visits (range 1-11). Eighty-six percent (n=74) experienced at least one seizure, most commonly generalized tonic-clonic, with mean age at onset of 9 years 7 months (SD 2y 10mo). Seizures were infrequent, typically occurring less often than once every 3 months, and were managed with one to two medications for most participants. Valproate (49%, n=36) and levetiracetam (41%, n=30) were the most commonly used seizure medications. Myoclonic seizures occurred infrequently (16%, n=14). Seizure severity did not vary by sex or genotype. Seizures showed mild worsening with increasing age.
INTERPRETATION: The neuronal ceroid lipofuscinoses (NCLs) represent a group of disorders unified by neurodegeneration and symptoms of blindness, seizures, motor impairment, and dementia. While NCLs are considered in the differential diagnosis of progressive myoclonus epilepsy, we show that myoclonic seizures are infrequent in JNCL. This highlights the NCLs as consisting of genetically distinct disorders with differing natural history.
© 2014 Mac Keith Press.

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Year:  2014        PMID: 25387857      PMCID: PMC4610252          DOI: 10.1111/dmcn.12634

Source DB:  PubMed          Journal:  Dev Med Child Neurol        ISSN: 0012-1622            Impact factor:   5.449


  20 in total

1.  Homogeneous polymerase chain reaction nucleobase quenching assay to detect the 1-kbp deletion in CLN3 that causes Batten disease.

Authors:  Paul G Rothberg; Denia Ramirez-Montealegre; Sharon D Frazier; David A Pearce
Journal:  J Mol Diagn       Date:  2004-08       Impact factor: 5.568

2.  Clinical and electrophysiological features of epilepsy in Italian patients with CLN8 mutations.

Authors:  Pasquale Striano; Nicola Specchio; Roberta Biancheri; Natalia Cannelli; Alessandro Simonati; Denise Cassandrini; Andrea Rossi; Claudio Bruno; Lucia Fusco; Roberto Gaggero; Federico Vigevano; Enrico Bertini; Federico Zara; Filippo M Santorelli; Salvatore Striano
Journal:  Epilepsy Behav       Date:  2006-11-28       Impact factor: 2.937

3.  A clinical rating scale for Batten disease: reliable and relevant for clinical trials.

Authors:  F J Marshall; E A de Blieck; J W Mink; L Dure; H Adams; S Messing; P G Rothberg; E Levy; T McDonough; J DeYoung; M Wang; D Ramirez-Montealegre; J M Kwon; D A Pearce
Journal:  Neurology       Date:  2005-07-26       Impact factor: 9.910

4.  Epilepsy and antiepileptic drug therapy in juvenile neuronal ceroid lipofuscinosis.

Authors:  L E Aberg; M Bäckman; E Kirveskari; P Santavuori
Journal:  Epilepsia       Date:  2000-10       Impact factor: 5.864

5.  Lamotrigine therapy in juvenile neuronal ceroid lipofuscinosis.

Authors:  L Aberg; E Kirveskari; P Santavuori
Journal:  Epilepsia       Date:  1999-06       Impact factor: 5.864

6.  Northern epilepsy: a novel form of neuronal ceroid-lipofuscinosis.

Authors:  R Herva; J Tyynelä; A Hirvasniemi; M Syrjäkallio-Ylitalo; M Haltia
Journal:  Brain Pathol       Date:  2000-04       Impact factor: 6.508

7.  Progressive myoclonic epilepsy: A clinical, electrophysiological and pathological study from South India.

Authors:  S Sinha; P Satishchandra; N Gayathri; T C Yasha; S K Shankar
Journal:  J Neurol Sci       Date:  2006-12-12       Impact factor: 3.181

8.  Isolation of a novel gene underlying Batten disease, CLN3. The International Batten Disease Consortium.

Authors: 
Journal:  Cell       Date:  1995-09-22       Impact factor: 41.582

Review 9.  Diagnosis of infantile spasms, Lennox-Gastaut syndrome, and progressive myoclonic epilepsy.

Authors:  W Donald Shields
Journal:  Epilepsia       Date:  2004       Impact factor: 5.864

10.  Neuronal ceroid lipofuscinosis: a clinicopathological study.

Authors:  Sanjib Sinha; P Satishchandra; Vani Santosh; N Gayatri; S K Shankar
Journal:  Seizure       Date:  2004-06       Impact factor: 3.184

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  10 in total

Review 1.  Future perspectives: Moving towards NCL treatments.

Authors:  Susan L Cotman; Sara E Mole; Romina Kohan
Journal:  Biochim Biophys Acta       Date:  2015-04-07

2.  Neuronal network dysfunction precedes storage and neurodegeneration in a lysosomal storage disorder.

Authors:  Rebecca C Ahrens-Nicklas; Luis Tecedor; Arron F Hall; Elena Lysenko; Akiva S Cohen; Beverly L Davidson; Eric D Marsh
Journal:  JCI Insight       Date:  2019-11-01

Review 3.  Drug Treatment of Progressive Myoclonic Epilepsy.

Authors:  Gregory L Holmes
Journal:  Paediatr Drugs       Date:  2020-04       Impact factor: 3.022

4.  A diagnostic confidence scheme for CLN3 disease.

Authors:  Margaux C Masten; Camille Corre; Alex R Paciorkowski; Amy Vierhile; Heather R Adams; Jennifer Vermilion; Grace A Zimmerman; Erika F Augustine; Jonathan W Mink
Journal:  J Inherit Metab Dis       Date:  2021-09-07       Impact factor: 4.750

Review 5.  Recent Insight into the Genetic Basis, Clinical Features, and Diagnostic Methods for Neuronal Ceroid Lipofuscinosis.

Authors:  Konrad Kaminiów; Sylwia Kozak; Justyna Paprocka
Journal:  Int J Mol Sci       Date:  2022-05-20       Impact factor: 6.208

6.  Non-convulsive Status Epilepticus in SEMA6B-Related Progressive Myoclonic Epilepsy: A Case Report With Literature Review.

Authors:  Jing Duan; Yan Chen; Zhanqi Hu; Yuanzhen Ye; Tian Zhang; Cong Li; Qi Zeng; Xia Zhao; Jiahui Mai; Yang Sun; Chao Liu; Wenxin Zheng; Yuhan Xiao; Jianxiang Liao; Li Chen
Journal:  Front Pediatr       Date:  2022-04-28       Impact factor: 3.569

7.  Astrocytes in juvenile neuronal ceroid lipofuscinosis (CLN3) display metabolic and calcium signaling abnormalities.

Authors:  Megan E Bosch; Tammy Kielian
Journal:  J Neurochem       Date:  2018-08-16       Impact factor: 5.372

8.  Seizure phenotype in CLN3 disease and its relation to other neurologic outcome measures.

Authors:  Myriam Abdennadher; Sara Inati; Ariane Soldatos; Gina Norato; Eva H Baker; Audrey Thurm; Luca Bartolini; Ruturaj Masvekar; William Theodore; Bibiana Bielekova; Forbes D Porter; An N Dang Do
Journal:  J Inherit Metab Dis       Date:  2021-02-15       Impact factor: 4.750

Review 9.  Juvenile neuronal ceroid lipofuscinosis (Batten disease): current insights.

Authors:  John R Ostergaard
Journal:  Degener Neurol Neuromuscul Dis       Date:  2016-08-01

Review 10.  Neuronal Ceroid Lipofuscinosis: The Multifaceted Approach to the Clinical Issues, an Overview.

Authors:  Alessandro Simonati; Ruth E Williams
Journal:  Front Neurol       Date:  2022-03-11       Impact factor: 4.003

  10 in total

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