Literature DB >> 21940967

Adverse effects of bone marrow stromal cell treatment of stroke in diabetic rats.

Jieli Chen1, Xinchun Ye, Tao Yan, Chunling Zhang, Xiao-Ping Yang, Xu Cui, Yishen Cui, Alex Zacharek, Cynthia Roberts, Xinfeng Liu, Xiangguo Dai, Mei Lu, Michael Chopp.   

Abstract

BACKGROUND AND
PURPOSE: Cell therapy with bone marrow stromal cells (BMSCs) improves functional recovery after stroke in nondiabetic rats. However, its effect on diabetics with stroke is unknown. This study investigated the effect of BMSCs on stroke outcome in Type 1 diabetic (T1DM) rats.
METHODS: T1DM was induced in adult male Wistar rats by injecting streptozotocin. Nondiabetic and T1DM rats were subjected to 2 hours of middle cerebral artery occlusion (MCAO), treated with or without BMSCs (3×10(6)) at 24 hours after MCAO, and monitored for 14 days.
RESULTS: Functional benefit was not detected in T1DM-MCAO treated with BMSC rats compared with corresponding T1DM-MCAO controls. BMSC treatment in T1DM-MCAO rats had increased mortality, blood-brain barrier leakage, brain hemorrhage, and angiogenesis. Internal carotid artery neointimal formation and cerebral arteriole narrowing/occlusion were also observed in T1DM-MCAO+BMSCs rats compared with T1DM-MCAO controls (P<0.05), but not in nondiabetic stroke rats. We further studied the underlying mechanisms responsible for BMSC-induced blood-brain barrier leakage and accelerated vascular damage in T1DM-MCAO rats. We found that the expression of angiogenin (an angiogenic factor) and ED1 (a marker for macrophages) was significantly increased in the T1DM-MCAO+BMSC rats in the ischemic brain and internal carotid artery compared with nontreated T1DM-MCAO rats, but not in nondiabetic stroke rats.
CONCLUSIONS: BMSC therapy in T1DM-MCAO rats does not improve functional outcome. On the contrary, it increases blood-brain barrier leakage and cerebral artery neointimal formation, and arteriosclerosis, which possibly is due to increased expression of angiogenin. Thus, BMSC treatment starting 24 hours after MCAO may not be beneficial for diabetic subjects with stroke.

Entities:  

Mesh:

Year:  2011        PMID: 21940967      PMCID: PMC3264886          DOI: 10.1161/STROKEAHA.111.627174

Source DB:  PubMed          Journal:  Stroke        ISSN: 0039-2499            Impact factor:   7.914


  25 in total

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2.  Angiopoietin/Tie2 pathway mediates type 2 diabetes induced vascular damage after cerebral stroke.

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Review 1.  Update on therapeutic mechanism for bone marrow stromal cells in ischemic stroke.

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Review 2.  Cell Therapy for Ischemic Stroke: How to Turn a Promising Preclinical Research into a Successful Clinical Story.

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4.  MiR-126 Contributes to Human Umbilical Cord Blood Cell-Induced Neurorestorative Effects After Stroke in Type-2 Diabetic Mice.

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Review 6.  Cell based therapies for ischemic stroke: from basic science to bedside.

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Review 8.  Hyperglycemia, acute ischemic stroke, and thrombolytic therapy.

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9.  Diabetes Worsens Functional Outcomes in Young Female Rats: Comparison of Stroke Models, Tissue Plasminogen Activator Effects, and Sexes.

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