Literature DB >> 21821327

Human masseter muscle fiber type properties, skeletal malocclusions, and muscle growth factor expression.

James Joseph Sciote1, Michael J Horton, Anthea M Rowlerson, Joel Ferri, John M Close, Gwenael Raoul.   

Abstract

PURPOSE: We identified masseter muscle fiber type property differences in subjects with dentofacial deformities. PATIENTS AND METHODS: Samples of masseter muscle were collected from 139 young adults during mandibular osteotomy procedures to assess mean fiber areas and percent tissue occupancies for the 4 fiber types that comprise the muscle. Subjects were classified into 1 of 6 malocclusion groups based on the presence of a skeletal Class II or III sagittal dimension malocclusion and either a skeletal open, deep, or normal bite vertical dimension malocclusion. In a subpopulation, relative quantities of the muscle growth factors IGF-I and GDF-8 gene expression were quantified by real-time polymerase chain reaction.
RESULTS: Fiber properties were not different in the sagittal malocclusion groups, but were very different in the vertical malocclusion groups (P ≤ .0004). There were significant mean fiber area differences for type II (P ≤ .0004) and type neonatal-atrial (P = .001) fiber types and for fiber percent occupancy differences for both type I-II hybrid fibers and type II fibers (P ≤ .0004). Growth factor expression differed by gender for IGF-I (P = .02) and GDF-8 (P < .01). The ratio of IGF-I:GDF-8 expression associates with type I and II mean fiber areas.
CONCLUSION: Fiber type properties are very closely associated with variations in vertical growth of the face, with statistical significance for overall comparisons at P ≤ .0004. An increase in masseter muscle type II fiber mean fiber areas and percent tissue occupancies is inversely related to increases in vertical facial dimension.
Copyright © 2012 American Association of Oral and Maxillofacial Surgeons. Published by Elsevier Inc. All rights reserved.

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Year:  2011        PMID: 21821327      PMCID: PMC3857112          DOI: 10.1016/j.joms.2011.04.007

Source DB:  PubMed          Journal:  J Oral Maxillofac Surg        ISSN: 0278-2391            Impact factor:   1.895


  46 in total

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  12 in total

1.  Nodal pathway genes are down-regulated in facial asymmetry.

Authors:  Romain Nicot; Molly Hottenstein; Gwenael Raoul; Joel Ferri; Michael Horton; John W Tobias; Elisabeth Barton; Patrick Gelé; James J Sciote
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Review 2.  Masseter function and skeletal malocclusion.

Authors:  J J Sciote; G Raoul; J Ferri; J Close; M J Horton; A Rowlerson
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4.  ACTN3 R577X genotypes associate with Class II and deepbite malocclusions.

Authors:  Brian Zebrick; Teesit Teeramongkolgul; Romain Nicot; Michael J Horton; Gwenael Raoul; Joel Ferri; Alexandre R Vieira; James J Sciote
Journal:  Am J Orthod Dentofacial Orthop       Date:  2014-10-28       Impact factor: 2.650

5.  Epigenetic influence of KAT6B and HDAC4 in the development of skeletal malocclusion.

Authors:  Ahrin Huh; Michael J Horton; Karen T Cuenco; Gwenael Raoul; Anthea M Rowlerson; Joel Ferri; James J Sciote
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6.  The Association of ACTN3 Rs1815739 Polymorphism with Various Malocclusion Phenotype.

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7.  Evaluation of factors related to morphological masseter muscle changes after preoperative orthodontic treatment in female patients with skeletal class III dentofacial deformities.

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8.  ACTN3 genotype influences masseter muscle characteristics and self-reported bruxism.

Authors:  Romain Nicot; Gwénaël Raoul; Alexandre R Vieira; Joël Ferri; James J Sciote
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9.  Molecular motor MYO1C, acetyltransferase KAT6B and osteogenetic transcription factor RUNX2 expression in human masseter muscle contributes to development of malocclusion.

Authors:  Heather Desh; S Lauren Gray; Michael J Horton; Gwenael Raoul; Anthea M Rowlerson; Joel Ferri; Alexandre R Vieira; James J Sciote
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