Literature DB >> 21745177

Focal thinning of the posterior corpus callosum: normal variant or post-traumatic?

Miriam H Beauchamp1, Michael Ditchfield, Cathy Catroppa, Michael Kean, Celia Godfrey, Jeffrey V Rosenfeld, Vicki Anderson.   

Abstract

AIM: The corpus callosum (CC) can be affected by childhood traumatic brain injury (TBI), through focal lesions, reductions in size and arrested development. Little is known, however, about what constitutes normal CC shape and appearance and how it may be affected in the long-term after early TBI.
METHODS: In this study, the appearance of the CC was investigated in individuals with TBI assessed 10 years post-injury (n = 52, mean age = 16.82 years, SD = 3.55 years, 24 male) and compared to age-matched healthy controls (n = 44, mean age = 15.77 years, SD = 1.21 years, 18 male). A simple visual analysis technique was used to code the appearance of the CC according to location of focal thinning and severity of thinning and results between groups were compared using Chi-square analysis.
RESULTS: A significantly higher proportion of patients with childhood TBI had focal posterior thinning of the CC than age-matched controls (p = 0.001).
CONCLUSIONS: The findings call into question previous conclusions that focal posterior thinning is a normal variant of development. Instead, attenuations of the posterior portion of the CC in the long-term may reflect altered cortical and callosal development as a result of early brain injury, although the functional significance of this remains to be determined.

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Year:  2011        PMID: 21745177     DOI: 10.3109/02699052.2011.589791

Source DB:  PubMed          Journal:  Brain Inj        ISSN: 0269-9052            Impact factor:   2.311


  6 in total

1.  Diffusion imaging of mild traumatic brain injury in the impact accelerated rodent model: A pilot study.

Authors:  Zora Kikinis; Marc Muehlmann; Ofer Pasternak; Sharon Peled; Praveen Kulkarni; Craig Ferris; Sylvain Bouix; Yogesh Rathi; Inga K Koerte; Steve Pieper; Alexander Yarmarkovich; Caryn L Porter; Bruce S Kristal; Martha E Shenton
Journal:  Brain Inj       Date:  2017-06-19       Impact factor: 2.311

Review 2.  GABAergic circuits of the basolateral amygdala and generation of anxiety after traumatic brain injury.

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Journal:  Amino Acids       Date:  2022-07-07       Impact factor: 3.789

3.  Theory of mind mediates the prospective relationship between abnormal social brain network morphology and chronic behavior problems after pediatric traumatic brain injury.

Authors:  Nicholas P Ryan; Cathy Catroppa; Richard Beare; Timothy J Silk; Louise Crossley; Miriam H Beauchamp; Keith Owen Yeates; Vicki A Anderson
Journal:  Soc Cogn Affect Neurosci       Date:  2016-01-21       Impact factor: 3.436

Review 4.  Is there evidence for neurodegenerative change following traumatic brain injury in children and youth? A scoping review.

Authors:  Michelle L Keightley; Katia J Sinopoli; Karen D Davis; David J Mikulis; Richard Wennberg; Maria C Tartaglia; Jen-Kai Chen; Charles H Tator
Journal:  Front Hum Neurosci       Date:  2014-03-19       Impact factor: 3.169

5.  Uncovering the neuroanatomical correlates of cognitive, affective and conative theory of mind in paediatric traumatic brain injury: a neural systems perspective.

Authors:  Nicholas P Ryan; Cathy Catroppa; Richard Beare; Timothy J Silk; Stephen J Hearps; Miriam H Beauchamp; Keith O Yeates; Vicki A Anderson
Journal:  Soc Cogn Affect Neurosci       Date:  2017-09-01       Impact factor: 3.436

6.  Traumatic brain injury, neuroimaging, and neurodegeneration.

Authors:  Erin D Bigler
Journal:  Front Hum Neurosci       Date:  2013-08-06       Impact factor: 3.169

  6 in total

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