Literature DB >> 21715151

The two faces of α- and γ-tocopherols: an in vitro and ex vivo investigation into VLDL, LDL and HDL oxidation.

Nida Nadeem1, Jayne V Woodside, Sean Kelly, Rachael Allister, Ian S Young, Jane McEneny.   

Abstract

BACKGROUND: Vitamin E and its derivatives, namely, the tocopherols, are known antioxidants, and numerous clinical trials have investigated their role in preventing cardiovascular disease; however, evidence to date remains inconclusive. Much of the in vitro research has focused on tocopherol's effects during low-density lipoprotein (LDL) oxidation, with little attention being paid to very LDL (VLDL) and high-density lipoprotein (HDL). Also, it is now becoming apparent that γ-tocopherol may potentially be more beneficial in relation to cardiovascular health.
OBJECTIVES: Do α- and γ-tocopherols become incorporated into VLDL, LDL and HDL and influence their oxidation potential in an in vitro and ex vivo situation?
DESIGN: Following (i) an in vitro investigation, where plasma was preincubated with increasing concentrations of either α- or γ-tocopherol and (ii) an in vivo 4-week placebo-controlled intervention with α- or γ-tocopherol. Tocopherol incorporation into VLDL, LDL and HDL was measured via high-pressure liquid chromatography, followed by an assessment of their oxidation potential by monitoring conjugated diene formation.
RESULTS: In vitro: Both tocopherols became incorporated into VLDL, LDL and HDL, which protected VLDL and LDL against oxidation. However and surprisingly, the incorporation into HDL demonstrated pro-oxidant properties. Ex vivo: Both tocopherols were incorporated into all three lipoproteins, protecting VLDL and LDL against oxidation; however, they enhanced the oxidation of HDL.
CONCLUSIONS: These results suggest that α- and γ-tocopherols display conflicting oxidant activities dependent on the lipoprotein being oxidized. Their pro-oxidant activity toward HDL may go some way to explain why supplementation studies with vitamin E have not been able to display cardioprotective effects.
Copyright © 2012 Elsevier Inc. All rights reserved.

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Year:  2011        PMID: 21715151     DOI: 10.1016/j.jnutbio.2011.04.009

Source DB:  PubMed          Journal:  J Nutr Biochem        ISSN: 0955-2863            Impact factor:   6.048


  7 in total

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Review 2.  Will Lipidation of ApoA1 through Interaction with ABCA1 at the Intestinal Level Affect the Protective Functions of HDL?

Authors:  Eric J Niesor
Journal:  Biology (Basel)       Date:  2015-01-06

Review 3.  Vitamin E, Turmeric and Saffron in Treatment of Alzheimer's Disease.

Authors:  Nur Adalier; Heath Parker
Journal:  Antioxidants (Basel)       Date:  2016-10-25

4.  Effects of glutathione s-transferase (GST) M1 and T1 polymorphisms on antioxidant vitamins and oxidative stress-related parameters in Korean subclinical hypertensive subjects after kale juice (Brassica oleracea acephala) supplementation.

Authors:  Hye-Jin Lee; Jeong-Hwa Han; Yoo Kyoung Park; Myung-Hee Kang
Journal:  Nutr Res Pract       Date:  2018-03-22       Impact factor: 1.926

5.  Protective Role of Natural and Semi-Synthetic Tocopherols on TNFα-Induced ROS Production and ICAM-1 and Cl-2 Expression in HT29 Intestinal Epithelial Cells.

Authors:  Vladana Domazetovic; Irene Falsetti; Caterina Viglianisi; Kristian Vasa; Cinzia Aurilia; Maria Stio; Stefano Menichetti; Teresa Iantomasi
Journal:  Antioxidants (Basel)       Date:  2021-01-22

6.  Biological effects of add-on eicosapentaenoic acid supplementation in diabetes mellitus and co-morbid depression: a randomized controlled trial.

Authors:  Roel J T Mocking; Johanna Assies; Mariska Bot; Eugene H J M Jansen; Aart H Schene; François Pouwer
Journal:  PLoS One       Date:  2012-11-28       Impact factor: 3.240

7.  Automated measurement method for the determination of vitamin E in plasma lipoprotein classes.

Authors:  Yuji Hirowatari; Hiroshi Yoshida; Hideo Kurosawa; Daisuke Manita; Norio Tada
Journal:  Sci Rep       Date:  2014-02-13       Impact factor: 4.379

  7 in total

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