Literature DB >> 21684136

A novel mechanism of coenzyme Q10 protects against human endothelial cells from oxidative stress-induced injury by modulating NO-related pathways.

Kun-Ling Tsai1, Yi-Hsiang Huang, Chung-Lan Kao, De-Ming Yang, Hsin-Chen Lee, Hsiang-Yun Chou, Yu-Chih Chen, Guang-Yuh Chiou, Li-Hsin Chen, Yi-Ping Yang, Tsan-Hung Chiu, Chiou-Sheng Tsai, Hsiu-Chung Ou, Shih-Hwa Chiou.   

Abstract

BACKGROUND: Atherosclerosis is a chronic inflammatory disease of the vessel wall associated with oxidized low-density lipoprotein (oxLDL)-induced apoptosis of endothelial cells. Coenzyme Q10 (CoQ10), a potent antioxidant and a critical intermediate of the electron transport chain, has been reported to inhibit LDL oxidation and thus the progression of atherosclerosis. However, its molecular mechanisms on endothelial cells remain still unclarified.
METHODS: In this study, primary human umbilical vein endothelial cell cultures treated with oxLDL were used to explore the protective effects of CoQ10.
RESULTS: Our results showed that CoQ10 attenuated the oxLDL-induced generation of reactive oxygen species and improved the antioxidant capacity. CoQ10 also attenuated the oxLDL-mediated down-regulation of endothelial nitric oxide synthase (eNOS) and up-regulation of inducible nitric oxide synthase (iNOS). In addition, CoQ10 suppressed oxLDL-activated NF-κB and downstream inflammatory mediators, including expression of adhesion molecules, release of proinflammatory cytokines and the adherence of monocytic THP-1 cells. Moreover, CoQ10 attenuated oxLDL-altered proapoptotic responses. The inhibitor of eNOS (L-NIO 10 μM) and iNOS (1400W 10 μM) as well as NO enhancer (SNP 10 μM) were used to clean up the mechanism.
CONCLUSION: These results provide new insight into the possible molecular mechanisms by which CoQ10 protects against atherogenesis by NO-related pathways.
Copyright © 2012 Elsevier Inc. All rights reserved.

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Year:  2011        PMID: 21684136     DOI: 10.1016/j.jnutbio.2011.01.011

Source DB:  PubMed          Journal:  J Nutr Biochem        ISSN: 0955-2863            Impact factor:   6.048


  36 in total

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Journal:  Indian Heart J       Date:  2018-06-08

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3.  Docetaxel Facilitates Endothelial Dysfunction through Oxidative Stress via Modulation of Protein Kinase C Beta: The Protective Effects of Sotrastaurin.

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Authors:  Mark Houston
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Review 7.  Nutraceuticals: Reviewing their Role in Chronic Disease Prevention and Management.

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Journal:  J Nat Med       Date:  2014-05-01       Impact factor: 2.343

9.  β3-adrenergic receptor activity modulates melanoma cell proliferation and survival through nitric oxide signaling.

Authors:  Massimo Dal Monte; Irene Fornaciari; Grazie Paola Nicchia; Maria Svelto; Giovanni Casini; Paola Bagnoli
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Review 10.  Roles of eNOS in atherosclerosis treatment.

Authors:  Fen-Fang Hong; Xiao-Yu Liang; Wei Liu; Sha Lv; Shu-Jin He; Hai-Bin Kuang; Shu-Long Yang
Journal:  Inflamm Res       Date:  2019-04-01       Impact factor: 4.575

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