Literature DB >> 21494847

Pharmacological inhibition of AKT sensitizes MCF-7 human breast cancer-initiating cells to radiation.

Jun-Fang Zhan1, Liang-Ping Wu, Long-Hua Chen, Ya-Wei Yuan, Guo-Zhu Xie, Ai-Min Sun, Ying Liu, Zhi-Xian Chen.   

Abstract

BACKGROUND: Caner-initiating cells (CICs or cancer stem cells) have been shown both experimentally and clinically to be resistant to radiation. The mechanism underlying radioresistance remains unclear.
METHODS: In the present study, we screened 51 genes which are potentially important in mediating radioresistance of breast CICs.
RESULTS: The expression of AKT1 and AKT2 at protein and mRNA levels was dramatically increased among the screened genes by 8 Gy radiation treatment in MCF-7 mammosphere cells (predominantly CD24(-/low)/CD44(+) CICs), but not in the bulk population of MCF-7 cells (predominantly CD24(+)/CD44(+)). Using apoptosis and clonogenic survival assays, we found pharmacological inhibition of AKT with selective inhibitors of AKT sensitized MCF-7 mammosphere cells, but not MCF-7 monolayer cells to radiation.
CONCLUSION: The present findings suggest that treatment with AKT inhibitors prior to ionizing radiation treatment may be a potential benefit to patients with breast cancer, in particular to eradiate breast CICs.

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Year:  2011        PMID: 21494847     DOI: 10.1007/s13402-011-0020-1

Source DB:  PubMed          Journal:  Cell Oncol (Dordr)        ISSN: 2211-3428            Impact factor:   6.730


  23 in total

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