Literature DB >> 21482944

Olfactory copy number association with age at onset of Alzheimer disease.

C A Shaw1, Y Li, J Wiszniewska, S Chasse, S N Y Zaidi, W Jin, B Dawson, K Wilhelmsen, J R Lupski, J W Belmont, R S Doody, K Szigeti.   

Abstract

OBJECTIVES: Copy number variants (CNVs) have been recognized as a source of genetic variation that contributes to disease phenotypes. Alzheimer disease (AD) has high heritability for occurrence and age at onset (AAO). We performed a cases-only genome-wide CNV association study for age at onset of AD.
METHODS: The discovery case series (n = 40 subjects with AD) was evaluated using array comparative genome hybridization (aCGH). A replication case series (n = 507 subjects with AD) was evaluated using Affymetrix array (n = 243) and multiplex ligation-dependent probe amplification (n = 264). Hazard models related onset age to CNV.
RESULTS: The discovery sample identified a chromosomal segment on 14q11.2 (19.3-19.5 Mb, NCBI build 36, UCSC hg18 March 2006) as a region of interest (genome-wide adjusted p = 0.032) for association with AAO of AD. This region encompasses a cluster of olfactory receptors. The replication sample confirmed the association (p = 0.035). The association was found for each APOE4 gene dosage (0, 1, and 2).
CONCLUSION: High copy number in the olfactory receptor region on 14q11.2 is associated with younger age at onset of AD.

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Year:  2011        PMID: 21482944      PMCID: PMC3090061          DOI: 10.1212/WNL.0b013e3182166df5

Source DB:  PubMed          Journal:  Neurology        ISSN: 0028-3878            Impact factor:   9.910


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