Literature DB >> 21438003

Increased level of serum Homocysteine in vitiligo.

Suman Singh1, Usha Singh, Shyam Sunder Pandey.   

Abstract

BACKGROUND: Vitiligo is an acquired depigmenting disorder caused by the destruction of melanocytes. The exact etiopathogenesis and mechanisms of vitiligo are not fully understood. Vitamin B12 and folic acid levels are decreased in vitiligo, which are the important cofactors required in the metabolism of Homocysteine (Hcy). Consequently, Hcy level increases in the circulation. Therefore, it is possible that increased Hcy plays a role in melanocytes destruction. The aim was to study for any association of vitiligo with serum Hcy level.
METHOD: A total of 30 vitiligo patients of both sexes and 30 control subjects were enrolled in this study. Sera from patients and controls were assayed for Hcy by Enzyme immunoassay. The collected data were analyzed by SPSS version-16.
RESULTS: The mean serum level of Hcy was significantly higher in patients with vitiligo as compared with healthy controls and its level was high in male patients as compared with female patients. The Hcy level in vegetarian patients was significantly higher as compared with nonvegetarian patients. The Hcy level was also significantly higher in active vitiligo patients as compared with stable vitiligo patients.
CONCLUSION: An increased serum Hcy may be a precipitating factor for vitiligo in the predisposed individuals. Serum Hcy is related to the gender of patients, activity of disease, and dietary habits of vitiligo patients.
© 2011 Wiley-Liss, Inc.

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Year:  2011        PMID: 21438003      PMCID: PMC6647724          DOI: 10.1002/jcla.20442

Source DB:  PubMed          Journal:  J Clin Lab Anal        ISSN: 0887-8013            Impact factor:   2.352


  13 in total

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5.  Cortisol and estradiol: nongenetic factors for hyperhomocyst(e)inemia.

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9.  Kinetics of total plasma homocysteine in subjects with hyperhomocysteinemia due to folate or cobalamin deficiency.

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Review 10.  [Hyperhomocysteinemia: an independent risk factor or a simple marker of vascular disease?. 1. Basic data].

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