Literature DB >> 21233592

Two- and 13-week inhalation toxicities of indium-tin oxide and indium oxide in rats.

Kasuke Nagano1, Kaoru Gotoh, Tatsuya Kasai, Shigetoshi Aiso, Tomoshi Nishizawa, Makoto Ohnishi, Naoki Ikawa, Yoko Eitaki, Kenichi Yamada, Heihachiro Arito, Shoji Fukushima.   

Abstract

OBJECTIVES: Two- and 13-week inhalation toxicities of indium-tin oxide (ITO) and indium oxide (IO) were characterized for risk assessments of workers exposed to ITO.
METHODS: F344 rats of both sexes were exposed by inhalation to ITO or IO aerosol for 6 h/day, 5 day/wk for 2 wk at 0, 0.1, 1, 10 or 100 mg/m(3) or 13 wk at 0, 0.1 or 1 mg/m(3). An aerosol generator and inhalation exposure system was constructed.
RESULTS: Blood and lung contents of indium were elevated in a dose-related manner in the ITO- and IO-exposed rats. ITO and IO particles were deposited in the lung, mediastinal lymph node and nasal-associated lymphoid tissue. Exposures to ITO and IO induced alveolar proteinosis, infiltrations of alveolar macrophages and inflammatory cells and alveolar epithelial hyperplasia in addition to increased lung weight. ITO affected the lung more severely than IO did. Fibrosis of alveolar wall developed and some of these lesions worsened at the end of the 26-week post-exposure period.
CONCLUSIONS: Persistent pulmonary lesions including alveolar proteinosis and macrophage infiltration occurred after 2- and 13-week inhalation exposures of rats to ITO and IO. Fibrosis of alveolar wall developed later. These lesions occurred after ITO exposure at the same concentration as the current occupational exposure limit in the USA and at blood indium levels below the biological exposure index in Japan for indium.

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Year:  2011        PMID: 21233592     DOI: 10.1539/joh.l10128

Source DB:  PubMed          Journal:  J Occup Health        ISSN: 1341-9145            Impact factor:   2.708


  11 in total

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3.  Application of the ICRP respiratory tract model to estimate pulmonary retention of industrially sampled indium-containing dusts.

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4.  Macrophage solubilization and cytotoxicity of indium-containing particles as in vitro correlates to pulmonary toxicity in vivo.

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5.  Use of and occupational exposure to indium in the United States.

Authors:  Cynthia J Hines; Jennifer L Roberts; Ronnee N Andrews; Matthew V Jackson; James A Deddens
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Authors:  Kristie Brock; Stacey E Anderson; Ewa Lukomska; Carrie Long; Katie Anderson; Nikki Marshall; B Jean Meade
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7.  Respirable indium exposures, plasma indium, and respiratory health among indium-tin oxide (ITO) workers.

Authors:  Kristin J Cummings; M Abbas Virji; Ji Young Park; Marcia L Stanton; Nicole T Edwards; Bruce C Trapnell; Brenna Carey; Aleksandr B Stefaniak; Kathleen Kreiss
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8.  Studies on the toxicity and distribution of indium compounds according to particle size in sprague-dawley rats.

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9.  Sintered indium-tin oxide particles induce pro-inflammatory responses in vitro, in part through inflammasome activation.

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10.  Pulmonary and Systemic Toxicity in a Rat Model of Pulmonary Alveolar Proteinosis Induced by Indium-Tin Oxide Nanoparticles.

Authors:  Nan Liu; Yi Guan; Chunling Zhou; Yongheng Wang; Zhanfei Ma; Sanqiao Yao
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