Literature DB >> 21228603

β-arrestin 2-mediated immune suppression induced by chronic stress.

Hui Li1, Dean Andrew Smalligan, Nanchang Xie, Avani Javer, Yi Zhang, Gregory Hanley, Deling Yin.   

Abstract

OBJECTIVE: Stress, either physical or psychological, can modulate immune function. However, the mechanisms associated with stress-induced immune suppression remain to be elucidated. β-Arrestin 2 serves as adaptor, scaffold, and/or signal transducer. The role of β-arrestin 2 in stress-induced immune suppression is not known yet. METHODS/
RESULTS: Here, we demonstrate that β-arrestin 2 deficiency in mice increases the sensitivity to the chronic stress-induced reduction in the number of splenocytes. Interestingly, the stress-induced suppression of T helper-type (Th) 1 cytokines and the increased production of Th2 cytokines were greatly enhanced in β-arrestin 2-deficient mice compared with wild-type mice. Moreover, inhibition of PI3K in β-arrestin 2-deficient mice exerts an additive effect on the stress-induced reduction in the number of splenocytes.
CONCLUSION: Our study demonstrates that a deficiency in β-arrestin 2 augments stress-induced immune suppression. 2011 S. Karger AG, Basel.

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Year:  2011        PMID: 21228603      PMCID: PMC3031150          DOI: 10.1159/000322868

Source DB:  PubMed          Journal:  Neuroimmunomodulation        ISSN: 1021-7401            Impact factor:   2.492


  44 in total

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