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Literature DB >> 2122465

Differentiation of F9 embryonal carcinoma cells induced by the c-jun and activated c-Ha-ras oncogenes.

Y Yamaguchi-Iwai¹, M Satake, Y Murakami, M Sakai, M Muramatsu, Y Ito.

Abstract

The activated c-Ha-ras oncogene induced AP1-site DNA-binding activity in F9 cells. This induction appeared to be due, at least in part, to the induction of c-jun transcription. Both activated c-Ha-ras and c-jun induced the differentiation of F9 cells to endoderm-like cells. Thus, AP1 appears to play a key role in the initial stage of F9 cell differentiation.

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Year: 1990 PMID： 2122465 PMCID： PMC55019 DOI： 10.1073/pnas.87.21.8670

Source DB: PubMed Journal: Proc Natl Acad Sci U S A ISSN： 0027-8424 Impact factor: 11.205

37 in total

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12 in total

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Journal: Biochem J Date: 1993-02-15 Impact factor: 3.857

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Authors: Esther Castellano; Eugenio Santos
Journal: Genes Cancer Date: 2011-03

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Journal: Mol Cell Biol Date: 2002-07 Impact factor: 4.272

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8. Activated Kras, but not Hras or Nras, may initiate tumors of endodermal origin via stem cell expansion.

Authors: Margaret P Quinlan; Steven E Quatela; Mark R Philips; Jeffrey Settleman
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Journal: EMBO J Date: 1992-01 Impact factor: 11.598