Literature DB >> 20962699

Contralateral suppression of otoacoustic emissions in tinnitus patients.

Leontien I Geven1, Emile de Kleine, Rolien H Free, Pim van Dijk.   

Abstract

OBJECTIVE: To compare the functioning of the medial olivocochlear efferent system between tinnitus patients and control subjects. STUDY
DESIGN: Prospective, nonrandomized controlled analysis of suppression of otoacoustic emissions with contralateral acoustic stimulation.
SETTING: Tertiary referral center. PATIENTS: Initial analysis of 97 tinnitus patients and 44 control subjects with click-evoked otoacoustic emission measurement. If subjects had reproducible otoacoustic emissions at 80 dB SPL, suppression of otoacoustic emission with contralateral acoustic stimulation was measured with a 65-dB click stimulus. This resulted in inclusion of 44 ears of tinnitus patients and 57 ears of control subjects. INTERVENTION: Suppression of the otoacoustic emissions generated by the 65-dB click stimulus was tested using contralateral broadband noise at 70 dB SPL. Suppression was calculated in half-octave frequency bands centered at 1.0, 1.4, 2.0, 2.8, and 4.0 kHz. MAIN OUTCOME MEASURE: The amount of suppression of the OAE, calculated in half-octave frequency bands.
RESULTS: Otoacoustic emission amplitudes were equal in both groups. Contralateral suppression of the signal was found in both patients and controls. The amount of suppression was equal, except for the 2.0- and 2.8-kHz frequency bands in the right ear (p value of 0.03, 0.008, respectively), for which the patients had less suppression.
CONCLUSION: The suppression of otoacoustic emissions with CAS seems equally effective in tinnitus patients and healthy controls. The minor differences between both groups suggest subtle differences in the function of the medial olivocochlear efferent system.

Entities:  

Mesh:

Year:  2011        PMID: 20962699     DOI: 10.1097/MAO.0b013e3181fcf180

Source DB:  PubMed          Journal:  Otol Neurotol        ISSN: 1531-7129            Impact factor:   2.311


  13 in total

1.  Increased contralateral suppression of otoacoustic emissions indicates a hyperresponsive medial olivocochlear system in humans with tinnitus and hyperacusis.

Authors:  Inge M Knudson; Christopher A Shera; Jennifer R Melcher
Journal:  J Neurophysiol       Date:  2014-09-17       Impact factor: 2.714

2.  Hyperactivity in the medial olivocochlear efferent system is a common feature of tinnitus and hyperacusis in humans.

Authors:  Joshua J Sturm; Catherine J C Weisz
Journal:  J Neurophysiol       Date:  2015-02-18       Impact factor: 2.714

3.  The Neural Bases of Tinnitus: Lessons from Deafness and Cochlear Implants.

Authors:  Marlies Knipper; Pim van Dijk; Holger Schulze; Birgit Mazurek; Patrick Krauss; Verena Scheper; Athanasia Warnecke; Winfried Schlee; Kerstin Schwabe; Wibke Singer; Christoph Braun; Paul H Delano; Andreas J Fallgatter; Ann-Christine Ehlis; Grant D Searchfield; Matthias H J Munk; David M Baguley; Lukas Rüttiger
Journal:  J Neurosci       Date:  2020-09-16       Impact factor: 6.167

4.  Noise-induced inner hair cell ribbon loss disturbs central arc mobilization: a novel molecular paradigm for understanding tinnitus.

Authors:  Wibke Singer; Annalisa Zuccotti; Mirko Jaumann; Sze Chim Lee; Rama Panford-Walsh; Hao Xiong; Ulrike Zimmermann; Christoph Franz; Hyun-Soon Geisler; Iris Köpschall; Karin Rohbock; Ksenya Varakina; Sandrine Verpoorten; Thomas Reinbothe; Thomas Schimmang; Lukas Rüttiger; Marlies Knipper
Journal:  Mol Neurobiol       Date:  2012-11-16       Impact factor: 5.590

5.  A connection between the Efferent Auditory System and Noise-Induced Tinnitus Generation. Reduced contralateral suppression of TEOAEs in patients with noise-induced tinnitus.

Authors:  Panagiota Lalaki; Stavros Hatzopoulos; Guiscardo Lorito; Krzysztof Kochanek; Lech Sliwa; Henryk Skarzynski
Journal:  Med Sci Monit       Date:  2011-07

Review 6.  The study of otoacoustic emissions and the suppression of otoacoustic emissions in subjects with tinnitus and normal hearing: an insight to tinnitus etiology.

Authors:  Lucieny Serra; Gabriela Novanta; Andre Lopes Sampaio; Carlos Augusto Oliveira; Ronaldo Granjeiro; Silvia Cristina Braga
Journal:  Int Arch Otorhinolaryngol       Date:  2014-06-18

Review 7.  Specific synaptopathies diversify brain responses and hearing disorders: you lose the gain from early life.

Authors:  Marlies Knipper; Rama Panford-Walsh; Wibke Singer; Lukas Rüttiger; Ulrike Zimmermann
Journal:  Cell Tissue Res       Date:  2015-04-07       Impact factor: 5.249

8.  Evaluation of distortion and transient evoked otoacoustic emission in tinnitus patients with normal hearing.

Authors:  Helnaz Mokrian; Abdolreza Shaibanizadeh; Saeid Farahani; Shohreh Jalaie; Parvane Mahdi; Amin Amali; Homa Arian Nahad
Journal:  Iran J Otorhinolaryngol       Date:  2014-01

9.  The reduced cochlear output and the failure to adapt the central auditory response causes tinnitus in noise exposed rats.

Authors:  Lukas Rüttiger; Wibke Singer; Rama Panford-Walsh; Masahiro Matsumoto; Sze Chim Lee; Annalisa Zuccotti; Ulrike Zimmermann; Mirko Jaumann; Karin Rohbock; Hao Xiong; Marlies Knipper
Journal:  PLoS One       Date:  2013-03-13       Impact factor: 3.240

10.  The Hyperactivity of Efferent Auditory System in Patients with Schizophrenia: A Transient Evoked Otoacoustic Emissions Study.

Authors:  Noor Alaudin Abdul Wahab; Suzaily Wahab; Abdul Hamid Abdul Rahman; Dinsuhaimi Sidek; Mohd Normani Zakaria
Journal:  Psychiatry Investig       Date:  2015-10-14       Impact factor: 2.505

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