Literature DB >> 20628232

A common founder mutation in the EDA-A1 gene in X-linked hypodontia.

Mazen Kurban1, Eleni Michailidis, Muhammad Wajid, Yutaka Shimomura, Angela M Christiano.   

Abstract

BACKGROUND: X-linked recessive hypohidrotic ectodermal dysplasia (XLHED; OMIM 305100) is a rare genodermatosis characterized clinically by developmental abnormalities affecting the teeth, hair and sweat glands. Mutations in the EDA-A1 gene have been associated with XLHED. Recently, mutations in the EDA-A1 gene have also been implicated in isolated X-linked recessive hypodontia (XLRH; OMIM 313500).
METHODS: We analyzed the DNA from members of 3 unrelated Pakistani families with XLRH for mutations in the EDA-A1 gene through direct sequencing and performed haplotype analysis.
RESULTS: We identified a common missense mutation in both families designated c.1091T→C (p.M364T). Haplotype analysis revealed that this is a founder mutation in the 3 families.
CONCLUSION: XLHED is a syndrome with variable clinical presentations that contain a spectrum of findings, including hypodontia. We suggest that XLRH should be grouped under XLHED as both share several phenotypic and genotypic similarities.

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Year:  2010        PMID: 20628232      PMCID: PMC2992807          DOI: 10.1159/000314329

Source DB:  PubMed          Journal:  Dermatology        ISSN: 1018-8665            Impact factor:   5.366


  18 in total

1.  Mutations leading to X-linked hypohidrotic ectodermal dysplasia affect three major functional domains in the tumor necrosis factor family member ectodysplasin-A.

Authors:  P Schneider; S L Street; O Gaide; S Hertig; A Tardivel; J Tschopp; L Runkel; K Alevizopoulos; B M Ferguson; J Zonana
Journal:  J Biol Chem       Date:  2001-03-14       Impact factor: 5.157

2.  X-linked anhidrotic (hypohidrotic) ectodermal dysplasia is caused by mutation in a novel transmembrane protein.

Authors:  J Kere; A K Srivastava; O Montonen; J Zonana; N Thomas; B Ferguson; F Munoz; D Morgan; A Clarke; P Baybayan; E Y Chen; S Ezer; U Saarialho-Kere; A de la Chapelle; D Schlessinger
Journal:  Nat Genet       Date:  1996-08       Impact factor: 38.330

3.  Ectodysplasin is released by proteolytic shedding and binds to the EDAR protein.

Authors:  O Elomaa; K Pulkkinen; U Hannelius; M Mikkola; U Saarialho-Kere; J Kere
Journal:  Hum Mol Genet       Date:  2001-04-15       Impact factor: 6.150

4.  Mutations in the human homologue of mouse dl cause autosomal recessive and dominant hypohidrotic ectodermal dysplasia.

Authors:  A W Monreal; B M Ferguson; D J Headon; S L Street; P A Overbeek; J Zonana
Journal:  Nat Genet       Date:  1999-08       Impact factor: 38.330

5.  The anhidrotic ectodermal dysplasia gene (EDA) undergoes alternative splicing and encodes ectodysplasin-A with deletion mutations in collagenous repeats.

Authors:  M Bayés; A J Hartung; S Ezer; J Pispa; I Thesleff; A K Srivastava; J Kere
Journal:  Hum Mol Genet       Date:  1998-10       Impact factor: 6.150

6.  Ectodysplasin is a collagenous trimeric type II membrane protein with a tumor necrosis factor-like domain and co-localizes with cytoskeletal structures at lateral and apical surfaces of cells.

Authors:  S Ezer; M Bayés; O Elomaa; D Schlessinger; J Kere
Journal:  Hum Mol Genet       Date:  1999-10       Impact factor: 6.150

7.  Functional analysis of Ectodysplasin-A mutations causing selective tooth agenesis.

Authors:  Gabriele Mues; Aubry Tardivel; Laure Willen; Hitesh Kapadia; Robyn Seaman; Sylvia Frazier-Bowers; Pascal Schneider; Rena N D'Souza
Journal:  Eur J Hum Genet       Date:  2010-01       Impact factor: 4.246

Review 8.  Ectodysplasin signaling in development.

Authors:  Marja L Mikkola; Irma Thesleff
Journal:  Cytokine Growth Factor Rev       Date:  2003 Jun-Aug       Impact factor: 7.638

9.  Gene defect in ectodermal dysplasia implicates a death domain adapter in development.

Authors:  D J Headon; S A Emmal; B M Ferguson; A S Tucker; M J Justice; P T Sharpe; J Zonana; P A Overbeek
Journal:  Nature       Date:  2001 Dec 20-27       Impact factor: 49.962

10.  Edar/Eda interactions regulate enamel knot formation in tooth morphogenesis.

Authors:  A S Tucker; D J Headon; P Schneider; B M Ferguson; P Overbeek; J Tschopp; P T Sharpe
Journal:  Development       Date:  2000-11       Impact factor: 6.868

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  3 in total

1.  Mutation of KREMEN1, a modulator of Wnt signaling, is responsible for ectodermal dysplasia including oligodontia in Palestinian families.

Authors:  Yasmin A Issa; Lara Kamal; Amal Abu Rayyan; Dima Dweik; Sarah Pierce; Ming K Lee; Mary-Claire King; Tom Walsh; Moien Kanaan
Journal:  Eur J Hum Genet       Date:  2016-04-06       Impact factor: 4.246

2.  A novel frameshift mutation in the EDA gene in an Iranian patient affected by X-linked hypohidrotic ectodermal dysplasia.

Authors:  Marzieh Rahbaran; Maryam Hassani Doabsari; Simindokht Salavitabar; Neda Mokhberian; Ziba Morovvati; Saeid Morovvati
Journal:  Cell Mol Biol Lett       Date:  2019-08-19       Impact factor: 5.787

3.  Deleterious Variants in WNT10A, EDAR, and EDA Causing Isolated and Syndromic Tooth Agenesis: A Structural Perspective from Molecular Dynamics Simulations.

Authors:  Asia Parveen; Sher Alam Khan; Muhammad Usman Mirza; Hina Bashir; Fatima Arshad; Maria Iqbal; Waseem Ahmad; Ahsan Wahab; Amal Fiaz; Sidra Naz; Fareeha Ashraf; Tayyaba Mobeen; Salman Aziz; Syed Shoaib Ahmed; Noor Muhammad; Nehal F Hassib; Mostafa I Mostafa; Nagwa E Gaboon; Roquyya Gul; Saadullah Khan; Matheus Froeyen; Muhammad Shoaib; Naveed Wasif
Journal:  Int J Mol Sci       Date:  2019-10-24       Impact factor: 5.923

  3 in total

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