Literature DB >> 20620233

Optimizing catecholaminergic polymorphic ventricular tachycardia therapy in calsequestrin-mutant mice.

Guy Katz1, Assad Khoury, Efrat Kurtzwald, Edith Hochhauser, Eyal Porat, Asher Shainberg, Jonathan G Seidman, Christine E Seidman, Abraham Lorber, Michael Eldar, Michael Arad.   

Abstract

BACKGROUND: Catecholaminergic polymorphic ventricular tachycardia (CPVT) is a lethal arrhythmia provoked by physical or emotional stress and mediated by spontaneous Ca(2+) release and delayed after-depolarizations. Beta-adrenergic blockers are the therapy of choice but fail to control arrhythmia in up to 50% of patients.
OBJECTIVE: To optimize antiarrhythmic therapy in recessively inherited CPVT caused by calsequestrin (CASQ2) mutations.
METHODS: Murine heart rhythm telemetry was obtained at rest, during treadmill exercise, and after injection of epinephrine. The protocol was repeated after injection of different antiarrhythmic drugs. Results were then validated in human patients.
RESULTS: Adult CASQ2 mutant mice had complex ventricular arrhythmia at rest and developed bidirectional and polymorphic ventricular tachycardia on exertion. Class I antiarrhythmic agents (procainamide, lidocaine, flecainide) were ineffective in controlling arrhythmia. Propranolol and sotalol attenuated arrhythmia at rest but failed to prevent VT during sympathetic stimulation. The calcium channel blocker verapamil showed a dose-dependent protection against CPVT. Verapamil was more effective than the dihydropyridine L-type Ca(2+) channel blocker nifedipine, and its activity was markedly enhanced when combined with propranolol. Human patients homozygous for CASQ2(D307H) mutation, remaining symptomatic despite chronic β-blocker therapy, underwent exercise testing according to the Bruce protocol with continuous electrocardiogram recording. Verapamil was combined with propranolol at maximum tolerated doses. Adding verapamil attenuated ventricular arrhythmia and prolonged exercise duration in five of 11 patients.
CONCLUSION: Verapamil is highly effective against catecholamine-induced arrhythmia in mice with CASQ2 mutations and may potentiate the antiarrhythmic activity of β-blockers in humans with CPVT2.
Copyright © 2010 Heart Rhythm Society. Published by Elsevier Inc. All rights reserved.

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Year:  2010        PMID: 20620233      PMCID: PMC4103178          DOI: 10.1016/j.hrthm.2010.07.004

Source DB:  PubMed          Journal:  Heart Rhythm        ISSN: 1547-5271            Impact factor:   6.343


  35 in total

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3.  Is the beta3-adrenergic receptor a new target for treatment of post-infarct ventricular tachyarrhythmias and prevention of sudden cardiac death?

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Journal:  Heart Rhythm       Date:  2007-11-01       Impact factor: 6.343

Review 4.  Catecholaminergic polymorphic ventricular tachycardia from bedside to bench and beyond.

Authors:  Guy Katz; Michael Arad; Michael Eldar
Journal:  Curr Probl Cardiol       Date:  2009-01       Impact factor: 5.200

5.  The role of nitrergic system in lidocaine-induced convulsion in the mouse.

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6.  Autosomal recessive catecholamine- or exercise-induced polymorphic ventricular tachycardia: clinical features and assignment of the disease gene to chromosome 1p13-21.

Authors:  H Lahat; M Eldar; E Levy-Nissenbaum; T Bahan; E Friedman; A Khoury; A Lorber; D L Kastner; B Goldman; E Pras
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8.  Calcium channel blockers and beta-blockers versus beta-blockers alone for preventing exercise-induced arrhythmias in catecholaminergic polymorphic ventricular tachycardia.

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9.  Left cardiac sympathetic denervation for catecholaminergic polymorphic ventricular tachycardia.

Authors:  Arthur A M Wilde; Zahurul A Bhuiyan; Lia Crotti; Mario Facchini; Gaetano M De Ferrari; Thomas Paul; Chiara Ferrandi; Dave R Koolbergen; Attilio Odero; Peter J Schwartz
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Review 10.  Mouse models of human arrhythmia syndromes.

Authors:  M J Killeen; G Thomas; I N Sabir; A A Grace; C L-H Huang
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  15 in total

1.  Inhibition of cardiac Ca2+ release channels (RyR2) determines efficacy of class I antiarrhythmic drugs in catecholaminergic polymorphic ventricular tachycardia.

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Journal:  Circ Arrhythm Electrophysiol       Date:  2011-01-26

2.  Power and pitfalls of using transgenic mice to optimize therapy for CPVT: a need for prospective placebo-controlled clinical trials in genetic arrhythmia disorders.

Authors:  Björn C Knollmann
Journal:  Heart Rhythm       Date:  2010-07-29       Impact factor: 6.343

3.  Alpha blockade potentiates CPVT therapy in calsequestrin-mutant mice.

Authors:  Efrat Kurtzwald-Josefson; Edith Hochhauser; Katia Bogachenko; Shiraz Harun-Khun; Guy Katz; Dan Aravot; Jonathan G Seidman; Christine E Seidman; Michael Eldar; Asher Shainberg; Michael Arad
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4.  The role of mutant protein level in autosomal recessive catecholamine dependent polymorphic ventricular tachycardia (CPVT2).

Authors:  Guy Katz; Asher Shainberg; Edith Hochhauser; Efrat Kurtzwald-Josefson; Ahuva Issac; Dalia El-Ani; Dan Aravot; Arnon Afek; Jonathan G Seidman; Christine E Seidman; Michael Eldar; Michael Arad
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5.  Catecholaminergic polymorphic ventricular tachycardia in children: analysis of therapeutic strategies and outcomes from an international multicenter registry.

Authors:  Thomas M Roston; Jeffrey M Vinocur; Kathleen R Maginot; Saira Mohammed; Jack C Salerno; Susan P Etheridge; Mitchell Cohen; Robert M Hamilton; Andreas Pflaumer; Ronald J Kanter; James E Potts; Martin J LaPage; Kathryn K Collins; Roman A Gebauer; Joel D Temple; Anjan S Batra; Christopher Erickson; Maria Miszczak-Knecht; Peter Kubuš; Yaniv Bar-Cohen; Michal Kantoch; Vincent C Thomas; Gabriele Hessling; Chris Anderson; Ming-Lon Young; Michel Cabrera Ortega; Yung R Lau; Christopher L Johnsrude; Anne Fournier; Prince J Kannankeril; Shubhayan Sanatani
Journal:  Circ Arrhythm Electrophysiol       Date:  2015-02-24

6.  Exercise training improves cardiac function and attenuates arrhythmia in CPVT mice.

Authors:  Efrat Kurtzwald-Josefson; Edith Hochhauser; Guy Katz; Eyal Porat; Jonathan G Seidman; Christine E Seidman; Yelena Chepurko; Asher Shainberg; Michael Eldar; Michael Arad
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7.  Effects of flecainide on exercise-induced ventricular arrhythmias and recurrences in genotype-negative patients with catecholaminergic polymorphic ventricular tachycardia.

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8.  [Catecholaminergic polymorphic ventricular tachycardia].

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