Literature DB >> 20615892

Inherited variations in AR, ESR1, and ESR2 genes are not associated with prostate cancer aggressiveness or with efficacy of androgen deprivation therapy.

Tong Sun1, Gwo-Shu Mary Lee, Lillian Werner, Mark Pomerantz, William K Oh, Philip W Kantoff, Matthew L Freedman.   

Abstract

BACKGROUND: Sex steroid hormone receptors mediate essential processes in normal prostate growth and contribute to prostate cancer development.
METHOD: In this study, we investigated the association between common inherited variation of the AR, ESR1, and ESR2 genes and two clinically relevant traits: the risk of developing aggressive prostate cancer and the response to androgen deprivation therapy (ADT) in a hospital-based cohort. A total of 43 tagging single nucleotide polymorphisms covering the loci of AR (n = 4), ESR1 (n = 32), and ESR2 (n = 7) were successfully genotyped in 4,073 prostate cancer cases.
RESULTS: None of these single nucleotide polymorphisms were significantly associated with disease aggressiveness as assessed by the D'Amico risk classification, pathologic stage, or the response to ADT.
CONCLUSIONS: Our results suggest that common genetic variations in AR, ESR1, or ESR2 are not strongly associated with prostate cancer aggressiveness or response to ADT. IMPACT: Our study did not find convincing evidence of inherited variations in the major receptors for androgens and estrogens and their associations with prostate cancer traits.

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Year:  2010        PMID: 20615892      PMCID: PMC3755451          DOI: 10.1158/1055-9965.EPI-10-0216

Source DB:  PubMed          Journal:  Cancer Epidemiol Biomarkers Prev        ISSN: 1055-9965            Impact factor:   4.254


  24 in total

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