Literature DB >> 20152363

Late hormonal function after testicular torsion.

Carmelo Romeo1, Pietro Impellizzeri, Teresa Arrigo, Pietro Antonuccio, Mariella Valenzise, Silvio Mirabelli, Francesca Astra Borruto, Gianfranco Scalfari, Francesco Arena, Filippo De Luca.   

Abstract

INTRODUCTION: Testicular torsion may be an important cause of male infertility. We aimed to investigate the late hormonal function in patients with testicular ischemia/reperfusion injury of the testis after orchidectomy or detorsion.
METHODS: Twenty patients (mean age, 13.6 years) were prospectively evaluated at a mean of 5 years after testicular torsion. The serum follicle-stimulating hormone, luteinizing hormone (before and after gonadotropin-releasing hormone stimulation), testosterone, and inhibin B were measured. Fifteen age-matched adolescents without evidence of endocrine disease were used as controls for inhibin B values. Data are quoted as mean +/- SEM.
RESULTS: Twelve patients were treated with detorsion and orchidopexy, and 8 underwent orchidectomy. Serum follicle-stimulating hormone, luteinizing hormone, and testosterone were all within the reference range. Inhibin B levels were significantly reduced in the 2 groups compared with the controls (34.5 +/- 5.2 vs 63.9 +/- 12.8 pg/mL, P = .02), but were not significantly different between the orchidectomy group and the group that underwent detorsion (41.3 +/- 9.7 vs 30.4 +/- 5.9 pg/mL, P = .41).
CONCLUSION: Hormonal testicular function can be compromised after testicular torsion, although the type of surgery (orchidectomy or orchidopexy) does not seem to change the effect of this ischemia/reperfusion injury. Copyright 2010 Elsevier Inc. All rights reserved.

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Year:  2010        PMID: 20152363     DOI: 10.1016/j.jpedsurg.2009.10.086

Source DB:  PubMed          Journal:  J Pediatr Surg        ISSN: 0022-3468            Impact factor:   2.545


  14 in total

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