Literature DB >> 19900227

Insulin resistance is not coupled with defective insulin secretion in primary hyperparathyroidism.

F Tassone1, M Procopio, L Gianotti, G Visconti, A Pia, M Terzolo, G Borretta.   

Abstract

AIMS: An increased frequency of both impaired glucose tolerance and Type 2 diabetes mellitus (DM) has been reported in primary hyperparathyroidism (pHPT), thus we sought to investigate insulin sensitivity and insulin secretion in a large series of pHPT patients. SUBJECTS AND METHODS: One hundred and twenty-two consecutive pHPT patients without known DM were investigated [age (mean +/- sd) 59.3 +/- 13.6 years, body mass index (BMI) 25.7 +/- 4.2 kg/m(2); serum calcium 2.8 +/- 0.25 mmol/l; PTH 203.2 +/- 145.4 ng/l]. Sixty-one control subjects were matched, according to the degree of glucose tolerance, in a 2 : 1 patient:control ratio. Fasting- and oral glucose tolerance test-derived estimates of insulin sensitivity and secretion were determined by means of the quantitative insulin sensitivity check index (QUICKI) and the insulin sensitivity index (ISI) composite.
RESULTS: Both the QUICKI and ISI composite were lower in pHPT patients than control subjects (P < 0.03 and P < 0.05, respectively) after adjusting for age, systolic blood pressure and BMI. Conversely, all insulin secretion estimates were significantly increased in pHPT patients than in control subjects (P < 0.04 and P < 0.03, respectively) and after adjusting for age, systolic blood pressure and BMI. Log serum calcium levels were negatively associated with the QUICKI and log ISI composite (R = -0.30, P = 0.001; R = -0.23, P = 0.020, respectively) in pHPT patients. Serum calcium levels significantly and independently contributed to impaired insulin sensitivity in multivariate analysis (QUICKI as dependent variable: beta = -0.31, P = 0.004, R(2) = 0.15; log ISI composite as dependent variable: beta = -0.29, P = 0.005, R(2) = 0.16).
CONCLUSIONS: Our study confirms a reduction in both basal and stimulated insulin sensitivity in primary hyperparathyroidism, in spite of increased insulin secretion. Moreover, our data show for the first time a significant relationship between hypercalcaemia and insulin sensitivity in this condition.

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Year:  2009        PMID: 19900227     DOI: 10.1111/j.1464-5491.2009.02804.x

Source DB:  PubMed          Journal:  Diabet Med        ISSN: 0742-3071            Impact factor:   4.359


  12 in total

1.  Insulin sensitivity in normocalcaemic primary hyperparathyroidism.

Authors:  F Tassone; M Maccario; L Gianotti; C Baffoni; M Pellegrino; S Cassibba; F Cesario; G Magro; G Borretta
Journal:  Endocrine       Date:  2013-09-25       Impact factor: 3.633

2.  Parathyroidectomy Decreases Insulin Resistance Index in Patients with Primary Hyperparathyroidism.

Authors:  Cevdet Duran; Barıs Sevinc; Orkide Kutlu; Omer Karahan
Journal:  Indian J Surg       Date:  2016-01-15       Impact factor: 0.656

3.  Prevalence and characteristics of metabolic syndrome in primary hyperparathyroidism.

Authors:  F Tassone; L Gianotti; C Baffoni; F Cesario; G Magro; M Pellegrino; I Emmolo; M Maccario; G Borretta
Journal:  J Endocrinol Invest       Date:  2011-12-21       Impact factor: 4.256

4.  The Association between Parathyroid Hormone Levels and the Cardiorenal Metabolic Syndrome in Non-Diabetic Chronic Kidney Disease.

Authors:  Georges Saab; Adam Whaley-Connell; Andrew Bombeck; Manjula Kurella Tamura; Suying Li; Shu-Cheng Chen; Samy I McFarlane; James R Sowers; Keith Norris; George L Bakris; Peter A McCullough
Journal:  Cardiorenal Med       Date:  2011-04-15       Impact factor: 2.041

5.  Investigation of insulin resistance in patients with normocalcemic primary hyperparathyroidism.

Authors:  Ilkay Cakir; Kursad Unluhizarci; Fatih Tanriverdi; Gulsah Elbuken; Zuleyha Karaca; Fahrettin Kelestimur
Journal:  Endocrine       Date:  2012-02-12       Impact factor: 3.633

6.  Analysis of 55 patients with multiple endocrine neoplasia type 1-associated insulinoma from a single center in China.

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Journal:  Orphanet J Rare Dis       Date:  2022-06-13       Impact factor: 4.303

7.  Cardiovascular risk and metabolic syndrome in primary hyperparathyroidism and their correlation to different clinical forms.

Authors:  M Procopio; M Barale; S Bertaina; S Sigrist; R Mazzetti; M Loiacono; G Mengozzi; E Ghigo; M Maccario
Journal:  Endocrine       Date:  2013-11-28       Impact factor: 3.633

8.  Primary hyperparathyroidism influences the expression of inflammatory and metabolic genes in adipose tissue.

Authors:  Monika H E Christensen; Simon N Dankel; Yngve Nordbø; Jan Erik Varhaug; Bjørg Almås; Ernst A Lien; Gunnar Mellgren
Journal:  PLoS One       Date:  2011-06-17       Impact factor: 3.240

Review 9.  Cardiorenal syndrome and vitamin D receptor activation in chronic kidney disease.

Authors:  Sirous Darabian; Manoch Rattanasompattikul; Parta Hatamizadeh; Suphamai Bunnapradist; Matthew J Budoff; Csaba P Kovesdy; Kamyar Kalantar-Zadeh
Journal:  Kidney Res Clin Pract       Date:  2012-01-18

10.  The association of 25 hydroxyvitamin D and parathyroid hormone with metabolic syndrome in two ethnic groups in South Africa.

Authors:  Jaya A George; Shane A Norris; Hendrik Emmanuel van Deventer; Nigel J Crowther
Journal:  PLoS One       Date:  2013-04-15       Impact factor: 3.240

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