Literature DB >> 19799605

Granulocyte-macrophage colony-stimulating factor enhances wound healing in diabetes via upregulation of proinflammatory cytokines.

Y Fang1, J Shen, M Yao, K W Beagley, B D Hambly, S Bao.   

Abstract

BACKGROUND: Chronic ulceration, especially in diabetes, remains a substantial clinical problem. Exogenous granulocyte-macrophage colony-stimulating factor (GM-CSF) is efficacious in the treatment of chronic wound healing in both animal models and patients, but its role in diabetic wounds remains to be explored. Objectives Using a diabetic mouse model, to investigate the role of GM-CSF in wound healing.
METHODS: Clinical observation, histopathology, immunohistochemistry and cytokine assays.
RESULTS: There was a significant reduction (50%) in GM-CSF production in the wounds of the diabetics compared with nondiabetics. Exogenous GM-CSF substantially enhanced the wound healing in diabetic mice, accompanied by increased interleukin-6 and monocyte chemoattractant protein-1 production. The elevated cytokines correlated with increased neovascularization, and infiltration of macrophages and neutrophils. GM-CSF showed no beneficial effects in nondiabetic wound healing.
CONCLUSIONS: Our results provide useful guidelines for the clinical management of chronic ulceration in diabetes.

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Year:  2009        PMID: 19799605     DOI: 10.1111/j.1365-2133.2009.09528.x

Source DB:  PubMed          Journal:  Br J Dermatol        ISSN: 0007-0963            Impact factor:   9.302


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