Literature DB >> 19795129

Impact of group IVA cytosolic phospholipase A2 gene polymorphisms on phenotypic features of patients with familial adenomatous polyposis.

Junji Umeno1, Takayuki Matsumoto, Motohiro Esaki, Yoji Kukita, Tomoko Tahira, Ritsuko Yanaru-Fujisawa, Shotaro Nakamura, Hisatomi Arima, Minako Hirahashi, Kenshi Hayashi, Mitsuo Iida.   

Abstract

OBJECTIVE: Group IVA cytosolic phospholipase A(2) (cPLA(2)alpha) plays a key role in tumorigenesis via generating arachidonic acids as the substrate of cyclooxygenase. The aim of this study was to elucidate the possible associations between cPLA ( 2 )alpha gene polymorphisms and phenotypic features of patients with familial adenomatous polyposis (FAP). PATIENTS AND METHODS: A tag single nucleotide polymorphisms (SNPs)-based genotype-phenotype association study of the cPLA ( 2 )alpha gene was conducted in 73 Japanese patients from 59 families with FAP. Based on the HapMap database, seven tag SNPs of the cPLA ( 2 )alpha gene were selected and genotyped by direct sequencing analysis. The genotype-phenotype association in relation to the adenomatous polyposis coli (APC) gene mutation was also assessed.
RESULTS: The single SNP analysis showed that rs3820185 C allele [odds ratio (OR), 2.5; 95% confidence interval (CI), 1.2-4.9] and rs127446200 GG genotype (OR, 10.9; 95%CI, 1.6-69.8), were more frequent in patients with gastric fundic gland polyposis (FGP) than in those without. Rs12749354 C allele was more frequently found in patients with small intestinal adenoma (OR, 7.0; 95% CI, 1.5-30.4; p = 0.008). This association was also significant when adjusted for covariates (age, sex, and APC mutation) in a logistic regression analysis (adjusted OR, 7.4; 95% CI, 1.2-64.2; p = 0.027).
CONCLUSIONS: The cPLA ( 2 )alpha gene may be a possible disease modifier gene in FAP.

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Year:  2009        PMID: 19795129     DOI: 10.1007/s00384-009-0808-x

Source DB:  PubMed          Journal:  Int J Colorectal Dis        ISSN: 0179-1958            Impact factor:   2.571


  40 in total

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2.  Singapore familial adenomatous polyposis (FAP) patients with classical adenomatous polyposis but undetectable APC mutations have accelerated cancer progression.

Authors:  Xia Cao; Yi Hong; Kong Weng Eu; Carol Loi; Peh Yean Cheah
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4.  Genetic analysis of an inherited predisposition to colon cancer in a family with a variable number of adenomatous polyps.

Authors:  M Leppert; R Burt; J P Hughes; W Samowitz; Y Nakamura; S Woodward; E Gardner; J M Lalouel; R White
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5.  Combined molecular and clinical approaches for the identification of families with familial adenomatous polyposis coli.

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6.  Impact of Phospholipase A2 group IIa gene polymorphism on phenotypic features of patients with familial adenomatous polyposis.

Authors:  Ritsuko Yanaru-Fujisawa; Takayuki Matsumoto; Yoji Kukita; Shotaro Nakamura; Takashi Yao; Kenshi Hayashi; Mitsuo Iida
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7.  Impact of Helicobacter pylori infection and mucosal atrophy on gastric lesions in patients with familial adenomatous polyposis.

Authors:  S Nakamura; T Matsumoto; Y Kobori; M Iida
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8.  Cytoprotection by prostaglandins in rats. Prevention of gastric necrosis produced by alcohol, HCl, NaOH, hypertonic NaCl, and thermal injury.

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Authors:  Takayuki Matsumoto; Motohiro Esaki; Ritsuko Yanaru-Fujisawa; Tomohiko Moriyama; Shinichiro Yada; Shotaro Nakamura; Takashi Yao; Mitsuo Iida
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10.  The secretory phospholipase A2 gene is a candidate for the Mom1 locus, a major modifier of ApcMin-induced intestinal neoplasia.

Authors:  M MacPhee; K P Chepenik; R A Liddell; K K Nelson; L D Siracusa; A M Buchberg
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3.  Group IVA Cytosolic Phospholipase A2 Regulates the G2-to-M Transition by Modulating the Activity of Tumor Suppressor SIRT2.

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4.  Two Chinese pedigrees for adenomatous polyposis coli: new mutations at codon 1309 and predisposition to phenotypic variations.

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5.  Targeting cytosolic phospholipase A2 α in colorectal cancer cells inhibits constitutively activated protein kinase B (AKT) and cell proliferation.

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  5 in total

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