| Literature DB >> 19692702 |
Christian Philipp Pallasch1, Michaela Patz, Yoon Jung Park, Susanne Hagist, Daniela Eggle, Rainer Claus, Svenja Debey-Pascher, Alexandra Schulz, Lukas P Frenzel, Julia Claasen, Nadine Kutsch, Günter Krause, Christine Mayr, Andreas Rosenwald, Christoph Plass, Joachim L Schultze, Michael Hallek, Clemens-Martin Wendtner.
Abstract
MicroRNAs (miRNA) play a key role in cellular regulation and, if deregulated, in the development of neoplastic disorders including chronic lymphocytic leukemia (CLL). RNAs from primary cells of 50 treatment-naive CLL patients and peripheral B cells of 14 healthy donors were applied to miRNA expression profiling using bead chip technology. In CLL cells, a set of 7 up- and 19 down-regulated miRNAs was identified. Among the miRNAs down-regulated in CLL cells, 6 of 10 miRNA promoters examined showed gain of methylation compared with normal B-cell controls. Subsequent target prediction of deregulated miRNAs revealed a highly significant binding prediction at the 3' untranslated region of the pleomorphic adenoma gene 1 (PLAG1) oncogene. Luciferase reporter assays including site-directed mutagenesis of binding sites revealed a significant regulation of PLAG1 by miR-181a, miR-181b, miR-107, and miR-424. Although expression of PLAG1 mRNA was not affected, PLAG1 protein expression was shown to be significantly elevated in CLL cells compared with the levels in healthy donor B cells. In summary, we could demonstrate disruption of miRNA-mediated translational control, partly due to epigenetic transcriptional silencing of miRNAs, with subsequent overexpression of the oncogenic transcription factor PLAG1 as a putative novel mechanism of CLL pathogenesis.Entities:
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Year: 2009 PMID: 19692702 PMCID: PMC2925729 DOI: 10.1182/blood-2009-06-229898
Source DB: PubMed Journal: Blood ISSN: 0006-4971 Impact factor: 22.113