Literature DB >> 19602159

Hypertrophic cardiomyopathy.

Jeffrey A Towbin1.   

Abstract

BACKGROUND: Hypertrophic cardiomyopathy (HCM) is one of the most common inherited cardiac disorders, with a prevalence in young adults of one in 500 and is defined by its wall thickening.
METHODS: This chapter of the supplement will present major clinical impacts of this disorder in its predilection to be inherited; its reputation as the most common cause of sudden death in young, healthy, athletic individuals; and its potential to develop heart failure due to either diastolic or systolic dysfunction, so-called "burned out" HCM. Underlying etiologies; diversity of morphologic, functional, and clinical features; and variable age of onset that differentiate the childhood from the adult form of disease will be discussed based on the literature and clinical experience.
RESULTS: In children less than 1 year of age, hypertrophy associated with systolic dysfunction is common. In contradistinction, among apparently healthy young adults, the prevalence of echcocardiographically defined HCM was reported to be as high as 0.2% and associated with diastolic dysfunction. In addition, overlaping disorders such as infiltrative and energy-dependent forms of HCM coexist with other atypical features in childhood, further confounding the presentations, treatments, and outcomes compared to adult disease.
CONCLUSION: HCM in childhood has a variety of etiologies which may influence diagnostic testing, treatments, and outcomes.

Entities:  

Mesh:

Year:  2009        PMID: 19602159     DOI: 10.1111/j.1540-8159.2009.02381.x

Source DB:  PubMed          Journal:  Pacing Clin Electrophysiol        ISSN: 0147-8389            Impact factor:   1.976


  8 in total

1.  Hypertrophic cardiomyopathy with cardiac rupture and tamponade caused by congenital disorder of glycosylation type Ia.

Authors:  Laura I Rudaks; Chad Andersen; T Y Khong; Andrew Kelly; Michael Fietz; Christopher P Barnett
Journal:  Pediatr Cardiol       Date:  2012-02-29       Impact factor: 1.655

Review 2.  Clinical applications of multiparametric CMR in left ventricular hypertrophy.

Authors:  Sabrina Nordin; Luke Dancy; James C Moon; Daniel M Sado
Journal:  Int J Cardiovasc Imaging       Date:  2018-03-03       Impact factor: 2.357

3.  Association of the Genetic Variation in the Long Non-Coding RNA FENDRR with the Risk of Developing Hypertrophic Cardiomyopathy.

Authors:  Elías Cuesta-Llavona; Rebeca Lorca; Valeria Rolle; Belén Alonso; Sara Iglesias; Julian Rodríguez-Reguero; Israel David Duarte-Herrera; Sergio Pérez-Oliveira; Alejandro Junco-Vicente; Claudia García Lago; Eliecer Coto; Juan Gómez
Journal:  Life (Basel)       Date:  2022-05-30

4.  Ablation of cardiac myosin binding protein-C disrupts the super-relaxed state of myosin in murine cardiomyocytes.

Authors:  James W McNamara; Amy Li; Nicola J Smith; Sean Lal; Robert M Graham; Kristina Bezold Kooiker; Sabine J van Dijk; Cristobal G Dos Remedios; Samantha P Harris; Roger Cooke
Journal:  J Mol Cell Cardiol       Date:  2016-03-26       Impact factor: 5.000

Review 5.  The diagnosis of hypertrophic cardiomyopathy by cardiovascular magnetic resonance.

Authors:  Radwa A Noureldin; Songtao Liu; Marcelo S Nacif; Daniel P Judge; Marc K Halushka; Theodore P Abraham; Carolyn Ho; David A Bluemke
Journal:  J Cardiovasc Magn Reson       Date:  2012-02-20       Impact factor: 5.364

6.  Private mitochondrial DNA variants in danish patients with hypertrophic cardiomyopathy.

Authors:  Christian M Hagen; Frederik H Aidt; Ole Havndrup; Paula L Hedley; Morten K Jensen; Jørgen K Kanters; Tam T Pham; Henning Bundgaard; Michael Christiansen
Journal:  PLoS One       Date:  2015-04-29       Impact factor: 3.240

7.  MYBPC3 mutations are associated with a reduced super-relaxed state in patients with hypertrophic cardiomyopathy.

Authors:  James W McNamara; Amy Li; Sean Lal; J Martijn Bos; Samantha P Harris; Jolanda van der Velden; Michael J Ackerman; Roger Cooke; Cristobal G Dos Remedios
Journal:  PLoS One       Date:  2017-06-28       Impact factor: 3.240

8.  Mitochondrial haplogroups modify the risk of developing hypertrophic cardiomyopathy in a Danish population.

Authors:  Christian M Hagen; Frederik H Aidt; Paula L Hedley; Morten K Jensen; Ole Havndrup; Jørgen K Kanters; Johanna C Moolman-Smook; Severin O Larsen; Henning Bundgaard; Michael Christiansen
Journal:  PLoS One       Date:  2013-08-05       Impact factor: 3.240

  8 in total

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