Literature DB >> 19596916

Toll-like receptor 4 activation increases Akt phosphorylation in colon cancer cells.

Hung Q Doan1, Kanika A Bowen, Lindsey A Jackson, B Mark Evers.   

Abstract

BACKGROUND: Toll-like receptors (TLRs) are involved in innate immunity. Overexpression of TLRs has been implicated in various types of cancer including colorectal cancer (CRC). The phosphatidylinositol-3'-kinase (PI3K)/Akt signaling pathway is involved in CRC growth and progression. In this study, we determined whether TLR4 signaling and PI3K/Akt pathway activation occur in CRCs.
MATERIALS AND METHODS: Human CRCs and adjacent mucosa were evaluated for TLR4 expression. CRC cell lines were treated with lipopolysaccharide (LPS), endogenous TLR4 ligand, to assess Akt phosphorylation.
RESULTS: Human CRCs overexpressed TLR4 compared to matched normal mucosa. Additionally, TLR4 was expressed in CRC cells and LPS treatment increased Akt phosphorylation of TLR4-positive CRCs in a time-dependent manner.
CONCLUSION: Our results identify TLR4 expression in human CRCs and activation of PI3K with LPS treatment. These findings suggest possible treatment strategies targeting TLR4 in CRC.

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Year:  2009        PMID: 19596916      PMCID: PMC2901163     

Source DB:  PubMed          Journal:  Anticancer Res        ISSN: 0250-7005            Impact factor:   2.480


  27 in total

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Authors:  Qingding Wang; Xiaofu Wang; Ambrosio Hernandez; Mark R Hellmich; Zoran Gatalica; B Mark Evers
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