Literature DB >> 19450994

The impact of Skp2 overexpression on recurrence-free survival following radical prostatectomy.

Paul L Nguyen1, Douglas I Lin, Junyi Lei, Michelangelo Fiorentino, Elke Mueller, Michael H Weinstein, Michele Pagano, Massimo Loda.   

Abstract

BACKGROUND: In several human cancers, overexpression of Skp2 (S-phase kinase associated protein 2), which targets p27 for degradation, portends a poorer prognosis. We examined whether Skp2 overexpression is associated with recurrence following radical prostatectomy (RP) for prostate cancer.
METHODS: Immunohistochemical staining for Skp2, p27, and MIB-1 was performed on 109 men with node-negative prostate cancer surgically managed from 1985-1996. Associations between the stains were tested and Cox regression was used to determine the association between Skp2 expression and time to biochemical recurrence following RP.
RESULTS: The 12 tumors (11%) with Skp2 overexpression all had correspondingly low p27 expression (P=0.006), and a similar inverse Skp2/p27 relationship was seen in vitro in LNCap cells. Skp2 overexpression in tissue was associated with higher Gleason score (P=0.002), more advanced pathological stage (P=0.01), and higher MIB-1 index (P=0.03), but a more favorable PSA profile (P=0.04). Five men received a TURP. Among 104 who received RP, median follow-up was 67 months (range: 0.2-218). After adjusting for PSA, pathologic stage, and Gleason score, Skp2 overexpression remained significantly associated with a shorter time to biochemical recurrence (adjusted hazard ratio 4.8 (95% C.I. 1.6-14, P=0.004)). The median time to recurrence with high vs. low Skp2 was 4 vs. 54 months.
CONCLUSIONS: Skp2 overexpression was seen in a significant minority of surgically-managed men and was independently associated with a higher risk of recurrence, raising the possibility that Skp2 could be useful as a prognostic biomarker and as a potential molecular target for novel systemic agents in prostate cancer.
Copyright © 2011 Elsevier Inc. All rights reserved.

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Year:  2009        PMID: 19450994      PMCID: PMC5437980          DOI: 10.1016/j.urolonc.2009.03.022

Source DB:  PubMed          Journal:  Urol Oncol        ISSN: 1078-1439            Impact factor:   3.498


  18 in total

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4.  Risk of prostate cancer-specific mortality following biochemical recurrence after radical prostatectomy.

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6.  Natural history of progression after PSA elevation following radical prostatectomy.

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7.  Androgen suppresses the proliferation of androgen receptor-positive castration-resistant prostate cancer cells via inhibition of Cdk2, CyclinA, and Skp2.

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8.  Skp2 deficiency restricts the progression and stem cell features of castration-resistant prostate cancer by destabilizing Twist.

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9.  Targeting of cytosolic phospholipase A2α impedes cell cycle re-entry of quiescent prostate cancer cells.

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10.  Flavokawain A induces deNEDDylation and Skp2 degradation leading to inhibition of tumorigenesis and cancer progression in the TRAMP transgenic mouse model.

Authors:  Xuesen Li; Noriko N Yokoyama; Saiyang Zhang; Lina Ding; Hong-min Liu; Michael B Lilly; Dan Mercola; Xiaolin Zi
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