Literature DB >> 19397476

Inhibition of HGF/MET as therapy for malignancy.

Sanjay Naran1, Xinglu Zhang, Steven J Hughes.   

Abstract

BACKGROUND: Inhibition of inappropriate tyrosine kinase activity by neoplasms is an attractive strategy for the treatment of malignancy.
OBJECTIVE: We aimed to produce a concise review of the potential role of hepatocyte growth factor (HGF)/Mesenchymal-epithelial transition factor (MET) tyrosine kinase pathway inhibition in the treatment of cancer.
METHODS: The current literature, abstracts and internet resources related to HGF/MET structure, function and inhibition are summarized. The potential of inhibiting this pathway as a therapy for cancer and remaining hurdles prior to routine clinical use of MET inhibition are discussed. RESULTS/
CONCLUSIONS: Current knowledge suggests that the inhibition of the HGF/MET pathway has significant potential for the treatment of cancer. A number of MET inhibitor molecules are nearing completion of their development for clinical use.

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Year:  2009        PMID: 19397476     DOI: 10.1517/14728220902853917

Source DB:  PubMed          Journal:  Expert Opin Ther Targets        ISSN: 1472-8222            Impact factor:   6.902


  14 in total

Review 1.  The role of cancer-associated myofibroblasts in intrahepatic cholangiocarcinoma.

Authors:  Alphonse E Sirica
Journal:  Nat Rev Gastroenterol Hepatol       Date:  2011-11-29       Impact factor: 46.802

Review 2.  Radioimmunotherapy of solid tumors: searching for the right target.

Authors:  Hong Song; George Sgouros
Journal:  Curr Drug Deliv       Date:  2011-01       Impact factor: 2.565

3.  Modulation of hepatocyte growth factor secretion in human female reproductive tract stromal fibroblasts by poly (I:C) and estradiol.

Authors:  Kimberly D Coleman; Mimi Ghosh; Sarah G Crist; Jacqueline A Wright; Richard M Rossoll; Charles R Wira; John V Fahey
Journal:  Am J Reprod Immunol       Date:  2011-08-23       Impact factor: 3.886

4.  Genomic abnormalities in invasive endocervical adenocarcinoma correlate with pattern of invasion: biologic and clinical implications.

Authors:  Anjelica Hodgson; Yutaka Amemiya; Arun Seth; Matthew Cesari; Bojana Djordjevic; Carlos Parra-Herran
Journal:  Mod Pathol       Date:  2017-07-21       Impact factor: 7.842

5.  (-)-Epigallocatechin-3-gallate inhibits Met signaling, proliferation, and invasiveness in human colon cancer cells.

Authors:  Christine A Larsen; Roderick H Dashwood
Journal:  Arch Biochem Biophys       Date:  2010-03-31       Impact factor: 4.013

6.  γ-Tocotrienol inhibits HGF-dependent mitogenesis and Met activation in highly malignant mammary tumour cells.

Authors:  N M Ayoub; S V Bachawal; P W Sylvester
Journal:  Cell Prolif       Date:  2011-10-04       Impact factor: 6.831

Review 7.  Tea catechins as inhibitors of receptor tyrosine kinases: mechanistic insights and human relevance.

Authors:  Christine A Larsen; Roderick H Dashwood; William H Bisson
Journal:  Pharmacol Res       Date:  2010-08-04       Impact factor: 7.658

8.  Tea catechins inhibit hepatocyte growth factor receptor (MET kinase) activity in human colon cancer cells: kinetic and molecular docking studies.

Authors:  Christine A Larsen; William H Bisson; Roderick H Dashwood
Journal:  J Med Chem       Date:  2009-11-12       Impact factor: 7.446

9.  c-Met activation through a novel pathway involving osteopontin mediates oncogenesis by the transcription factor LSF.

Authors:  Byoung Kwon Yoo; Rachel Gredler; Dong Chen; Prasanna K Santhekadur; Paul B Fisher; Devanand Sarkar
Journal:  J Hepatol       Date:  2011-04-13       Impact factor: 25.083

Review 10.  The possible diagnostic and prognostic use of systemic chemokine profiles in clinical medicine—the experience in acute myeloid leukemia from disease development and diagnosis via conventional chemotherapy to allogeneic stem cell transplantation.

Authors:  Håkon Reikvam; Hanne Fredly; Astrid Olsnes Kittang; Oystein Bruserud
Journal:  Toxins (Basel)       Date:  2013-02-18       Impact factor: 4.546

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