Literature DB >> 19392979

Suppression of Mamu-AG by RNA interference.

Jessica G Drenzek1, Jolanta Vidiguriene, Geminis Vidiguris, Richard L Grendell, Svetlana V Dambaeva, Maureen Durning, Thaddeus G Golos.   

Abstract

PROBLEM: The role of placental major histocompatibility complex (MHC) class I molecules in pregnancy is not well understood. Mamu-AG, the rhesus monkey homology of human leukocyte antigen (HLA)-G expressed in the human placenta, was targeted for degradation by RNA interference (RNAi), a powerful tool to aid in determining gene function, to determine the effect that this knockdown has on NK cell function. METHOD OF STUDY: A series of potential target short hairpin RNA (shRNA) sequences to suppress Mamu-AG expression was screened, which identified an optimal sequence to use in transfection experiments. Knockdown in two different Mamu-AG-expressing cell lines was measured by flow cytometry. Cytotoxicity assays were performed to correlate Mamu-AG expression with NK cell cytotoxicity.
RESULTS: Decreased expression of Mamu-AG by short interfering RNA (siRNA) (70-80%) in cell types tested was associated with increased lysis of Mamu-AG target cells.
CONCLUSION: Target sequences have been identified that knocked down Mamu-AG expression by RNAi and increased lysis by NK cells. This supports the concept that NK cell receptors recognize this placental non-classical MHC class I molecule.
© 2009 John Wiley & Sons A/S.

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Year:  2009        PMID: 19392979      PMCID: PMC2889004          DOI: 10.1111/j.1600-0897.2009.00706.x

Source DB:  PubMed          Journal:  Am J Reprod Immunol        ISSN: 1046-7408            Impact factor:   3.886


  24 in total

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5.  Identification of a novel MHC class I gene, Mamu-AG, expressed in the placenta of a primate with an inactivated G locus.

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