Literature DB >> 19337198

Mechanisms of primary and secondary resistance to imatinib in chronic myeloid leukemia.

Alfonso Quintás-Cardama1, Hagop M Kantarjian, Jorge E Cortes.   

Abstract

BACKGROUND: Although the vast majority of patients with chronic myeloid leukemia (CML) respond to the tyrosine kinase inhibitor (TKI) imatinib mesylate, resistance might occur de novo or during treatment.
METHODS: The authors reviewed the known mechanisms of primary and secondary resistance to imatinib and other TKIs used in the management of CML.
RESULTS: Mutations within the kinase domain of BCR-ABLI account for 30% to 40% of cases of imatinib resistance. Other mechanisms include BCR-ABLI amplification, overexpression of the SRC family of kinases, and pharmacokinetic and pharmacodynamic factors.
CONCLUSIONS: Although not all resistance mechanisms have been identified and understood, several agents based on the known mechanisms have already been designed and developed and are beginning clinical trials.

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Year:  2009        PMID: 19337198     DOI: 10.1177/107327480901600204

Source DB:  PubMed          Journal:  Cancer Control        ISSN: 1073-2748            Impact factor:   3.302


  80 in total

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