Literature DB >> 19226523

A strategy for identifying circulating placental RNA markers for fetal growth assessment.

Winnie W I Pang1, Michelle H Y Tsui, Daljit Sahota, Tak Y Leung, Tze K Lau, Y M Dennis Lo, Rossa W K Chiu.   

Abstract

OBJECTIVE: To evaluate whether circulating placental mRNAs in maternal plasma could serve as markers for the assessment of fetal growth or intrauterine growth restriction (IUGR).
METHODS: From a panel of placental transcripts detectable in maternal plasma identified by microarray previously, we chose growth-related transcripts, namely CSH1, GH2, KISS1, and ADAM12, as potential growth markers. Relationships between the maternal plasma mRNA concentrations with several fetal growth indicators were studied. Maternal plasma mRNA concentrations from IUGR pregnancies with or without pre-eclampsia (PET) were compared with gestational age matched controls cross-sectionally and longitudinally. The four transcripts were quantified by one-step real-time RT-PCR.
RESULTS: Maternal plasma GH2 mRNA significantly correlated with birth weight and fetal biometric measurements. Maternal plasma ADAM12 mRNA concentration was significantly higher in IUGR with PET than normal pregnancies in the cross-sectional comparison. No significant difference was observed for all markers between IUGR without PET and controls in both the cross-sectional and longitudinal comparisons.
CONCLUSION: This study presents a potential strategy in identifying surrogate markers for the study of fetal growth. Circulating GH2 mRNA in maternal plasma appeared to be associated with fetal growth. The utility of this strategy and the currently assessed markers could be explored in further studies. (c) 2009 John Wiley & Sons, Ltd.

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Year:  2009        PMID: 19226523     DOI: 10.1002/pd.2230

Source DB:  PubMed          Journal:  Prenat Diagn        ISSN: 0197-3851            Impact factor:   3.050


  13 in total

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Review 2.  Biosensors for Detection of Human Placental Pathologies: A Review of Emerging Technologies and Current Trends.

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3.  Differential Expression of HrtA1 and ADAM12 in Placentas from Preeclamptic and Normotensive Pregnancies.

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4.  Integrative single-cell and cell-free plasma RNA transcriptomics elucidates placental cellular dynamics.

Authors:  Jason C H Tsang; Joaquim S L Vong; Lu Ji; Liona C Y Poon; Peiyong Jiang; Kathy O Lui; Yun-Bi Ni; Ka Fai To; Yvonne K Y Cheng; Rossa W K Chiu; Yuk Ming Dennis Lo
Journal:  Proc Natl Acad Sci U S A       Date:  2017-08-22       Impact factor: 11.205

Review 5.  Current, Emerging, and Future Applications of Digital PCR in Non-Invasive Prenatal Diagnosis.

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Journal:  Mol Diagn Ther       Date:  2018-04       Impact factor: 4.074

6.  Intrauterine growth retardation-associated syncytin b hypermethylation in maternal rat blood revealed by DNA methylation array analysis.

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7.  Chorioamnionitis Occurring in Women With Preterm Rupture of the Fetal Membranes Is Associated With a Dynamic Increase in mRNAs Coding Cytokines in the Maternal Circulation.

Authors:  Owen Stock; Lavinia Gordon; Jada Kapoor; Susan P Walker; Clare Whitehead; Tu'uhevaha J Kaitu'u-Lino; Gabrielle Pell; Natalie J Hannan; Stephen Tong
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8.  The levels of hypoxia-regulated microRNAs in plasma of pregnant women with fetal growth restriction.

Authors:  J-F Mouillet; T Chu; C A Hubel; D M Nelson; W T Parks; Y Sadovsky
Journal:  Placenta       Date:  2010-07-29       Impact factor: 3.481

9.  Prediction of Fetal Growth Restriction by Analyzing the Messenger RNAs of Angiogenic Factor in the Plasma of Pregnant Women.

Authors:  Shin Takenaka; Walter Ventura; Anna Freni Sterrantino; Akihiro Kawashima; Keiko Koide; Kyoko Hori; Antonio Farina; Akihiko Sekizawa
Journal:  Reprod Sci       Date:  2014-12-09       Impact factor: 3.060

10.  Identification of messenger RNA of fetoplacental source in maternal plasma of women with normal pregnancies and pregnancies with intrauterine growth restriction.

Authors:  Paola Ayala Ramírez; Reggie García Robles; Juan Diego Rojas; Martha Bermúdez; Jaime Bernal
Journal:  Colomb Med (Cali)       Date:  2012-09-30
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