Literature DB >> 19137409

A comparison of sodium bicarbonate infusion versus normal saline infusion and its combination with oral acetazolamide for prevention of contrast-induced nephropathy: a randomized, double-blind trial.

Maryam Pakfetrat1, Mohammad Hossein Nikoo, Leila Malekmakan, Mahmood Tabandeh, Jamshid Roozbeh, Mahshid Hashemi Nasab, Mohammad Ali Ostovan, Soheila Salari, Mohammad Kafi, Najmeh Moin Vaziri, Farzad Adl, Mehdi Hosseini, Parviz Khajehdehi.   

Abstract

BACKGROUND: Contrast-induced nephropathy (CIN) is commonly encountered. Because the therapy of choice for prevention of CIN is controversial, in this study we compared the preventive efficacy of bicarbonate (Bi) infusion in dextrose water versus normal saline (NLS) infusion alone or in combination with oral acetazolamide (AZ).
METHODS: In a double-blind and randomized clinical trial, all patients undergoing coronary angiography or percutaneous coronary intervention received NLS (NLS group), its combination with AZ (AZ group) or infusion of Bi (Bi group) before the procedures. RIFLE (risk of renal failure, injury to the kidney, failure of kidney function, loss of kidney function, and end-stage renal disease) criteria were used to define CIN-associated acute kidney injury (AKI).
RESULTS: The risk of AKI in CIN was significantly lower in the Bi and AZ groups than in the NLS group (P <or= 0.04). Comparing pre-treatment and post-treatment values in each group the following results were obtained: serum creatinine (Scr) increased and eGFR decreased significantly in the NLS group (P = 0.04) and in all patients (P = 0.001, P = 0.02, respectively). In addition, serum potassium decreased significantly in the Bi and NLS groups (P <or= 0.02). Also, serum Bi increased significantly in the Bi group (P = 0.001) whereas it decreased significantly in the AZ group (P = 0.001). Urinary pH also increased in all groups (P <or= 0.04) except the NLS group (P > 0.05).
CONCLUSIONS: It seems that both Bi and AZ reduce the risk of CIN-related AKI, and close monitoring of serum potassium is needed during bicarbonate infusion.

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Year:  2009        PMID: 19137409     DOI: 10.1007/s11255-008-9520-y

Source DB:  PubMed          Journal:  Int Urol Nephrol        ISSN: 0301-1623            Impact factor:   2.370


  33 in total

1.  Assessing kidney function--measured and estimated glomerular filtration rate.

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3.  Life-threatening ventricular arrhythmia induced by hypokalemia during sodium bicarbonate infusion.

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4.  Effect of sodium bicarbonate administration on blood constituents of horses.

Authors:  L J Rivas; K W Hinchcliff; C W Kohn; R A Sams; D J Chew
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Review 5.  Contrast material-induced renal failure: an overview.

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6.  The role of endothelin in radiocontrast nephropathy.

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7.  [Contrast-induced nephropathy and acute renal failure following emergent cardiac catheterization: incidence, risk factors and prognosis].

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10.  Frequency of serum creatinine changes in the absence of iodinated contrast material: implications for studies of contrast nephrotoxicity.

Authors:  Jeffrey H Newhouse; David Kho; Qasim A Rao; Justin Starren
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  27 in total

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Review 3.  Acute kidney injury.

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Journal:  BMJ Clin Evid       Date:  2011-03-28

4.  Risk of nephropathy after consumption of nonionic contrast media by children undergoing cardiac angiography: a prospective study.

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5.  Impact of heart failure on the incidence of contrast-induced nephropathy in patients with chronic kidney disease.

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Review 7.  Acute kidney injury in the elderly population.

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Review 8.  Sodium bicarbonate infusion for prevention of acute kidney injury: no evidence for superior benefit, but risk for harm?

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Review 9.  Prevention of contrast-induced AKI: a review of published trials and the design of the prevention of serious adverse events following angiography (PRESERVE) trial.

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Review 10.  Preventing contrast-induced nephropathy: problems, challenges and future directions.

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