Literature DB >> 18834226

CpG island methylator phenotype in colorectal cancers: comparison of the new and classic CpG island methylator phenotype marker panels.

Sun Lee1, Nam-Yun Cho, Eun Joo Yoo, Jung Ho Kim, Gyeong Hoon Kang.   

Abstract

CONTEXT: CpG island methylator phenotype (CIMP) designates a subset of colorectal cancers featuring concordant hypermethylation of multiple promoter CpG islands. Little is known about the clinical outcome or histologic characteristics of CIMP-positive colorectal cancers defined by recently identified CpG island methylator phenotype panels.
OBJECTIVE: To investigate and compare the molecular and clinicopathologic features of CIMP-positive colorectal cancers defined by classic (p16, hMLH1, MINT1, MINT2, MINT31) and new (CACNA1G, IGF2, NEUROG1, RUNX3, SOCS1) CIMP panels.
DESIGN: We analyzed 130 colorectal cancers for hypermethylation of both panels using methylation-specific polymerase chain reaction.
RESULTS: With at least 2 markers methylated, both classic (39/130; 23.1%) and new (23.1%) CIMP-positive colorectal cancers were significantly associated with proximal tumor location, microsatellite instability, and BRAF mutation (all P values were less than .05). The new panel outperformed the classic panel in detecting these features. With at least 3 markers methylated, new CIMP-positive colorectal cancers (16.9%) were closely associated with proximal tumor location, low frequency of KRAS mutation, and high frequency of BRAF mutation (all P values were less than .05), whereas classic CIMP-positive colorectal cancers (18.5%) were closely associated with proximal tumor location, frequent microsatellite instability, and frequent BRAF mutation (all P values were less than .05). Analyzing a combination of CIMP and microsatellite instability status, CIMP-positive/microsatellite instability-negative colorectal cancers had the worst clinical outcomes.
CONCLUSIONS: Whereas the classic panel outperformed in predicting clinical outcome, the new panel was superior in detecting known clinicopathologic features of CIMP but inferior in prognostication power.

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Year:  2008        PMID: 18834226     DOI: 10.5858/2008-132-1657-CIMPIC

Source DB:  PubMed          Journal:  Arch Pathol Lab Med        ISSN: 0003-9985            Impact factor:   5.534


  27 in total

1.  Differential clinicopathological features in microsatellite instability-positive colorectal cancers depending on CIMP status.

Authors:  Jeong Mo Bae; Mi Jung Kim; Jung Ho Kim; Jae Moon Koh; Nam-Yun Cho; Tae-You Kim; Gyeong Hoon Kang
Journal:  Virchows Arch       Date:  2011-04-15       Impact factor: 4.064

2.  Association of HPV with genetic and epigenetic alterations in colorectal adenocarcinoma from Indian population.

Authors:  Ruhina S Laskar; Fazlur R Talukdar; Javed H Choudhury; Seram Anil Singh; Sharbadeb Kundu; Bishal Dhar; Rosy Mondal; Sankar Kumar Ghosh
Journal:  Tumour Biol       Date:  2015-02-03

3.  Prognostic significance of hMLH1/hMSH2 gene mutations and hMLH1 promoter methylation in sporadic colorectal cancer.

Authors:  Yibaina Wang; Dandan Li; Xia Li; Chong Teng; Lin Zhu; Binbin Cui; Yashuang Zhao; Fulan Hu
Journal:  Med Oncol       Date:  2014-06-11       Impact factor: 3.064

4.  Correlation between smoking history and molecular pathways in sporadic colorectal cancer: a meta-analysis.

Authors:  Ke Chen; Guanggai Xia; Changhua Zhang; Yunwei Sun
Journal:  Int J Clin Exp Med       Date:  2015-03-15

5.  Aberrant DNA methylation of WNT pathway genes in the development and progression of CIMP-negative colorectal cancer.

Authors:  Orsolya Galamb; Alexandra Kalmár; Bálint Péterfia; István Csabai; András Bodor; Dezső Ribli; Tibor Krenács; Árpád V Patai; Barnabás Wichmann; Barbara Kinga Barták; Kinga Tóth; Gábor Valcz; Sándor Spisák; Zsolt Tulassay; Béla Molnár
Journal:  Epigenetics       Date:  2016-05-31       Impact factor: 4.528

Review 6.  Serrated neoplasia-role in colorectal carcinogenesis and clinical implications.

Authors:  Joep E G IJspeert; Louis Vermeulen; Gerrit A Meijer; Evelien Dekker
Journal:  Nat Rev Gastroenterol Hepatol       Date:  2015-05-12       Impact factor: 46.802

7.  Reduced FHIT expression is associated with mismatch repair deficient and high CpG island methylator phenotype colorectal cancer.

Authors:  Rabeah Abbas Al-Temaimi; Sindhu Jacob; Waleed Al-Ali; Diana Ann Thomas; Fahd Al-Mulla
Journal:  J Histochem Cytochem       Date:  2013-06-24       Impact factor: 2.479

8.  Epigenetic inactivation of the novel candidate tumor suppressor gene ITIH5 in colon cancer predicts unfavorable overall survival in the CpG island methylator phenotype.

Authors:  Vera Kloten; Michael Rose; Sophie Kaspar; Saskia von Stillfried; Ruth Knüchel; Edgar Dahl
Journal:  Epigenetics       Date:  2014-08-04       Impact factor: 4.528

9.  Associations between colorectal cancer molecular markers and pathways with clinicopathologic features in older women.

Authors:  N Jewel Samadder; Robert A Vierkant; Lori S Tillmans; Alice H Wang; Daniel J Weisenberger; Peter W Laird; Charles F Lynch; Kristin E Anderson; Amy J French; Robert W Haile; John D Potter; Susan L Slager; Thomas C Smyrk; Stephen N Thibodeau; James R Cerhan; Paul J Limburg
Journal:  Gastroenterology       Date:  2013-05-07       Impact factor: 22.682

10.  Prognostic implications of CpG island hypermethylator phenotype in colorectal cancers.

Authors:  Jung Ho Kim; So Hyun Shin; Hyeong Ju Kwon; Nam Yun Cho; Gyeong Hoon Kang
Journal:  Virchows Arch       Date:  2009-11-13       Impact factor: 4.064

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