Literature DB >> 18803085

Risk factors and mechanisms of anaphylactoid reactions to acetylcysteine in acetaminophen overdose.

Nasrin Pakravan1, W Stephen Waring, Sushma Sharma, Christopher Ludlam, Ian Megson, D Nicholas Bateman.   

Abstract

BACKGROUND: Adverse effects to N-acetylcysteine (NAC) are well recognized, but their etiology and incidence are unclear.
METHODS: The nature and severity of adverse effects were prospectively studied in 169 patients and potential reaction mediators studied in 22 patients.
RESULTS: Adverse effects were minimal in 101 (59.8%), moderate in 51 (30.2%), and severe in 17 (10.1%). Features were nausea (70.4%), vomiting (60.4%), flushing (24.9%), pruritus (20.1%), dyspnea (13.6%), chest pain (7.1%), dizziness (7.7%), fever (4.7%), wheeze and bronchospasm (7.1%), and rash and urticaria (3.6%). Serum acetaminophen concentration was lower in patients with severe adverse effects: median (IQR) 46 mg/L (0 to 101 mg/L), moderate 108 mg/L (54 to 178 mg/L), and minimal 119 mg/L (77 to 174 mg/L), p = 0.002. Family history of allergy and female gender were independent risk factors for adverse effects. Severity of adverse effects was associated with histamine release: AUC for change from baseline histamine was -6 ng/mL min (-60 to 11 ng/mL min) in the minimal group, 26 ng/mL min (3-129 ng/mL min) in the moderate group, and 49 ng/mL min (21-68 ng/mL min) in the severe group (p = 0.01). There was no increase in tryptase and no differences between groups for NAC concentrations or hemostatic and inflammatory variables (factors II, VII, IX, X, vWF, tPA, IL6, and CRP).
CONCLUSION: Severity of adverse effects correlates with the extent of histamine release. Histamine release appears independent of tryptase suggesting a non-mast cell source. Acetaminophen is protective against adverse effects of NAC, and mechanisms by which acetaminophen might lessen histamine release require further attention.

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Year:  2008        PMID: 18803085     DOI: 10.1080/15563650802245497

Source DB:  PubMed          Journal:  Clin Toxicol (Phila)        ISSN: 1556-3650            Impact factor:   4.467


  24 in total

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2.  miRNA-324, a potential therapeutic target for paracetamol-induced liver injury.

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3.  An evaluation of the role of mixing techniques in the observed variation in acetylcysteine infusion concentrations.

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Review 4.  Novel Therapeutic Approaches Against Acetaminophen-induced Liver Injury and Acute Liver Failure.

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5.  Delays during the administration of acetylcysteine for the treatment of paracetamol overdose.

Authors:  George P Bailey; Javad Najafi; Muhammad E M O Elamin; W Stephen Waring; Simon H L Thomas; John R H Archer; David M Wood; Paul I Dargan
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6.  Anaphylactoid Reactions to Intravenous N-Acetylcysteine during Treatment for Acetaminophen Poisoning.

Authors:  Mark Yarema; Puja Chopra; Marco L A Sivilotti; David Johnson; Alberto Nettel-Aguirre; Benoit Bailey; Charlemaigne Victorino; Sophie Gosselin; Roy Purssell; Margaret Thompson; Daniel Spyker; Barry Rumack
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7.  An assessment of the variation in the concentration of acetylcysteine in infusions for the treatment of paracetamol overdose.

Authors:  George P Bailey; David M Wood; John R H Archer; Edmund Rab; Robert J Flanagan; Paul I Dargan
Journal:  Br J Clin Pharmacol       Date:  2016-09-29       Impact factor: 4.335

Review 8.  Paracetamol poisoning: beyond the nomogram.

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Review 9.  Evidence for the changing regimens of acetylcysteine.

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Review 10.  Novel acetylcysteine regimens for treatment of paracetamol overdose.

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Journal:  Ther Adv Drug Saf       Date:  2012-12
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