Literature DB >> 18751705

Dual targeting of EGFR and HER-2 in colon cancer cell lines.

Efstathia Giannopoulou1, Anna Antonacopoulou, Konstantina Floratou, Athanasios G Papavassiliou, Haralabos P Kalofonos.   

Abstract

PURPOSE: A number of studies have revealed that coexpression of EGFR and HER-2 has been found in a subset of colon cancers and may cooperatively promote tumor cell growth and survival. In the present work, two tyrosine kinase inhibitors, gefitinib and lapatinib, together with trastuzumab, raised a monoclonal antibody against HER-2 were evaluated in two colon cancer cell lines, DLD-1 and Caco-2. The aim of the study was to investigate their effect on tumor cell proliferation and apoptosis.
METHODS: Cell proliferation was assessed using the MTT assay and apoptosis was evaluated by DNA fragmentation and the Annexin V binding assay. EGFR and HER-2 protein and mRNA levels were evaluated by immunoblotting and quantitative RT-PCR, respectively.
RESULTS: Treatment of cells with each agent alone resulted in inhibition of cell proliferation after 48 h in a dose-dependent manner except for trastuzumab, which did not alter cell proliferation of DLD-1. Apoptosis increased in DLD-1 cells, after 24 h treatment with gefitinib. None of the tested agents altered apoptosis in Caco-2 cells. HER-2 and EGFR protein levels did not follow the changes of mRNA levels after treatment with the tested agents.
CONCLUSIONS: Tauhe inhibitory effect of these agents on cell proliferation and the induction of apoptosis differ for the two colon cancer cell lines under consideration. Further studies are necessary to investigate the way they exert their antitumor effect.

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Year:  2008        PMID: 18751705     DOI: 10.1007/s00280-008-0820-9

Source DB:  PubMed          Journal:  Cancer Chemother Pharmacol        ISSN: 0344-5704            Impact factor:   3.333


  6 in total

1.  In vitro combined treatment with cetuximab and trastuzumab inhibits growth of colon cancer cells.

Authors:  T Luca; V Barresi; G Privitera; N Musso; M Caruso; D F Condorelli; S Castorina
Journal:  Cell Prolif       Date:  2014-08-08       Impact factor: 6.831

2.  The dual EGFR/HER2 inhibitor lapatinib synergistically enhances the antitumor activity of the histone deacetylase inhibitor panobinostat in colorectal cancer models.

Authors:  Melissa J LaBonte; Peter M Wilson; Will Fazzone; Jared Russell; Stan G Louie; Anthony El-Khoueiry; Heinz-Josef Lenz; Robert D Ladner
Journal:  Cancer Res       Date:  2011-04-04       Impact factor: 12.701

3.  An in vitro study, evaluating the effect of sunitinib and/or lapatinib on two glioma cell lines.

Authors:  Efstathia Giannopoulou; Konstantinos Dimitropoulos; Andreas A Argyriou; Angelos K Koutras; Fotinos Dimitrakopoulos; Haralabos P Kalofonos
Journal:  Invest New Drugs       Date:  2009-07-15       Impact factor: 3.850

4.  Antiproliferative effect of exemestane in lung cancer cells.

Authors:  Angelos Koutras; Efstathia Giannopoulou; Ismini Kritikou; Anna Antonacopoulou; T R Jeffry Evans; Athanasios G Papavassiliou; Haralabos Kalofonos
Journal:  Mol Cancer       Date:  2009-11-24       Impact factor: 27.401

5.  Combination of gefitinib and DNA methylation inhibitor decitabine exerts synergistic anti-cancer activity in colon cancer cells.

Authors:  Yun-feng Lou; Zheng-zhi Zou; Pin-jia Chen; Guo-bin Huang; Bin Li; De-qing Zheng; Xiu-rong Yu; Xiao-yong Luo
Journal:  PLoS One       Date:  2014-05-29       Impact factor: 3.240

6.  Lapatinib-capecitabine versus capecitabine alone as radiosensitizers in RAS wild-type resectable rectal cancer, an adaptive randomized phase II trial (LaRRC trial): study protocol for a randomized controlled trial.

Authors:  Nuno Sousa; Olga Sousa; Lúcio Lara Santos; Rui Henrique; Manuel R Teixeira; Mário Dinis-Ribeiro; Armando Teixeira-Pinto
Journal:  Trials       Date:  2016-09-21       Impact factor: 2.279

  6 in total

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