Literature DB >> 18512489

Differential profile of the OPG/RANKL/RANK-system in degenerative aortic native and bioprosthetic valves.

Martin Steinmetz1, Dirk Skowasch, Nicolas Wernert, Ulrich Welsch, Clauss J Preusse, Armin Welz, Georg Nickenig, Gerhard Bauriedel.   

Abstract

BACKGROUND AND AIM OF THE STUDY: Although degenerative calcific aortic valve stenosis is the most common valvular disease among the elderly, neither the etiology underlying the condition nor degeneration of the bioprostheses is yet fully understood. The study aim was to assess the expression profile of those OPG/RANKL/RANK-system determinants known to act as key regulators of bone metabolism and the immune system in calcific aortic valve stenosis and porcine aortic bioprostheses.
METHODS: Valve probes from a total of 69 patients (41 with end-stage aortic stenosis, 11 with mild-to-moderate aortic sclerosis, 17 with degenerative porcine aortic bioprostheses) were explanted either during surgery or at autopsy. The presence and localization of OPG, RANKL, RANK and NF-kappaB were analyzed by immunostaining and morphometry.
RESULTS: The majority of stenotic and sclerotic valves exhibited cell-bound signals of OPG, RANKL, RANK and NF-kappaB, while bioprostheses showed only sparse signaling. As key findings, the percentage of cells labeled by OPG, RANK and NF-kappaB was increased in sclerotic valves compared with stenotic valves (each p < 0.001), whereas the frequency of RANKL was higher in stenotic compared to sclerotic valves (p < 0.001). As a consequence, the OPG/RANKL ratio was decreased in stenotic (0.83) compared to sclerotic valves (20.2).
CONCLUSION: The differential expression profile of specific members of the OPG/RANKL/RANK axis suggests an involvement of their determinants in native valve calcification, but not in the degeneration of porcine bioprostheses. Thus, these mediators of bone homeostasis may represent new targets for a more specified prevention and/or therapy of native aortic stenosis.

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Year:  2008        PMID: 18512489

Source DB:  PubMed          Journal:  J Heart Valve Dis        ISSN: 0966-8519


  12 in total

1.  The stretch responsive microRNA miR-148a-3p is a novel repressor of IKBKB, NF-κB signaling, and inflammatory gene expression in human aortic valve cells.

Authors:  Vishal Patel; Katrina Carrion; Andrew Hollands; Andrew Hinton; Thomas Gallegos; Jeffrey Dyo; Roman Sasik; Emma Leire; Gary Hardiman; Salah A Mohamed; Sanjay Nigam; Charles C King; Victor Nizet; Vishal Nigam
Journal:  FASEB J       Date:  2015-01-28       Impact factor: 5.191

Review 2.  Calcific aortic valve stenosis: methods, models, and mechanisms.

Authors:  Jordan D Miller; Robert M Weiss; Donald D Heistad
Journal:  Circ Res       Date:  2011-05-27       Impact factor: 17.367

3.  VEGF and RANKL regulation of NFATc1 in heart valve development.

Authors:  Michelle D Combs; Katherine E Yutzey
Journal:  Circ Res       Date:  2009-08-06       Impact factor: 17.367

Review 4.  Vascular calcification: an update on mechanisms and challenges in treatment.

Authors:  Meiting Wu; Cameron Rementer; Cecilia M Giachelli
Journal:  Calcif Tissue Int       Date:  2013-03-01       Impact factor: 4.333

5.  RANKL Expression Is Increased in Circulating Mononuclear Cells of Patients with Calcific Aortic Stenosis.

Authors:  Marcello Rattazzi; Elisabetta Faggin; Elisa Bertacco; Roberta Buso; Massimo Puato; Mario Plebani; Martina Zaninotto; Davide Condotta; Giacomo Zoppellaro; Leopoldo Pagliani; Giuseppe Tarantini; Sabino Iliceto; Elisa Covolo; Giuseppe Faggian; Francesco Onorati; Mikhail Dodonov; Alessandro Daniotti; Paola Pantano; Zoran Olivari; Giovanni Benfari; Paolo Pauletto
Journal:  J Cardiovasc Transl Res       Date:  2018-05-17       Impact factor: 4.132

6.  Effect of Bushen Huoxue decoction on inhibiting osteogenic differentiation of vascular smooth cells by regulating OPG/RANK/RANKL system in vascular calcification.

Authors:  Shi-Yi Liu; Xiang-Fei Meng; Shi-Wei Liu; Cong-Li Hao; Lan-Fang Li; Ning Zhang
Journal:  Ann Transl Med       Date:  2019-03

Review 7.  Vascular calcification and renal bone disorders.

Authors:  Kuo-Cheng Lu; Chia-Chao Wu; Jen-Fen Yen; Wen-Chih Liu
Journal:  ScientificWorldJournal       Date:  2014-07-17

8.  Sleep-disordered breathing is associated with depletion of circulating endothelial progenitor cells and elevation in pulmonary arterial pressure in patients with decompensated systolic heart failure.

Authors:  Han Zhang; Liu Feng; Qi-Lin Wan; Yan Hong; Yan-Ming Li; Guan-Chang Cheng; Xin-Qiang Han
Journal:  J Geriatr Cardiol       Date:  2015-07       Impact factor: 3.327

9.  The role of TNF-α and TNF superfamily members in the pathogenesis of calcific aortic valvular disease.

Authors:  Antonella Galeone; Domenico Paparella; Silvia Colucci; Maria Grano; Giacomina Brunetti
Journal:  ScientificWorldJournal       Date:  2013-11-06

Review 10.  The living aortic valve: From molecules to function.

Authors:  Adrian H Chester; Ismail El-Hamamsy; Jonathan T Butcher; Najma Latif; Sergio Bertazzo; Magdi H Yacoub
Journal:  Glob Cardiol Sci Pract       Date:  2014-01-29
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